Sarah E. Kobylewski, Ph.D.
Affiliations: | 2012 | Molecular Toxicology 0368 | University of California, Los Angeles, Los Angeles, CA |
Area:
Toxicology, Public HealthGoogle:
"Sarah Kobylewski"Cross-listing: Chemistry Tree
Parents
Sign in to add mentor| Curtis D. Eckhert | grad student | 2012 | UCLA (Chemistry Tree) | |
| (Boric Acid Causes ER Stress and Activates the eIF2alpha/ATF4 and ATF6 Branches of the Unfolded Protein Response in Prostate Cancer Cells and Using Toxicology in the Public Interest.) | ||||
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Publications
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| Kobylewski SE, Henderson KA, Yamada KE, et al. (2016) Activation of the EIF2α/ATF4 and ATF6 Pathways in DU-145 Cells by Boric Acid at the Concentration Reported in Men at the US Mean Boron Intake. Biological Trace Element Research |
| Henderson KA, Kobylewski SE, Yamada KE, et al. (2015) Boric acid induces cytoplasmic stress granule formation, eIF2α phosphorylation, and ATF4 in prostate DU-145 cells. Biometals : An International Journal On the Role of Metal Ions in Biology, Biochemistry, and Medicine. 28: 133-41 |
| Kobylewski SE, Henderson KA, Eckhert CD. (2012) Identification of ryanodine receptor isoforms in prostate DU-145, LNCaP, and PWR-1E cells. Biochemical and Biophysical Research Communications. 425: 431-5 |
| Henderson K, Stella SL, Kobylewski S, et al. (2009) Receptor activated Ca(2+) release is inhibited by boric acid in prostate cancer cells. Plos One. 4: e6009 |