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Toumy Guettouche, Ph.D. - Publications

Affiliations: 
2002 University of Miami, Coral Gables, FL 
Area:
Molecular Biology
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6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2010 Harashima A, Guettouche T, Barber GN. Phosphorylation of the NFAR proteins by the dsRNA-dependent protein kinase PKR constitutes a novel mechanism of translational regulation and cellular defense. Genes & Development. 24: 2640-53. PMID 21123651 DOI: 10.1101/Gad.1965010  0.418
2010 Harashima A, Guettouche T, Barber GN. PS2-64 Phosphorylations of the NFAR proteins constitute a novel, conserved mechanism of translational regelation and cellular defence Cytokine. 52: 63-64. DOI: 10.1016/J.Cyto.2010.07.269  0.345
2005 Guettouche T, Boellmann F, Lane WS, Voellmy R. Analysis of phosphorylation of human heat shock factor 1 in cells experiencing a stress. Bmc Biochemistry. 6: 4. PMID 15760475 DOI: 10.1186/1471-2091-6-4  0.732
2004 Boellmann F, Guettouche T, Guo Y, Fenna M, Mnayer L, Voellmy R. DAXX interacts with heat shock factor 1 during stress activation and enhances its transcriptional activity. Proceedings of the National Academy of Sciences of the United States of America. 101: 4100-5. PMID 15016915 DOI: 10.1073/Pnas.0304768101  0.659
2001 Guo Y, Guettouche T, Fenna M, Boellmann F, Pratt WB, Toft DO, Smith DF, Voellmy R. Evidence for a mechanism of repression of heat shock factor 1 transcriptional activity by a multichaperone complex. The Journal of Biological Chemistry. 276: 45791-9. PMID 11583998 DOI: 10.1074/Jbc.M105931200  0.652
1998 Zou J, Guo Y, Guettouche T, Smith DF, Voellmy R. Repression of heat shock transcription factor HSF1 activation by HSP90 (HSP90 complex) that forms a stress-sensitive complex with HSF1. Cell. 94: 471-80. PMID 9727490 DOI: 10.1016/S0092-8674(00)81588-3  0.781
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