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Calcagno DM, Chu A, Gaul S, et al. (2022) NOD-like receptor protein 3 activation causes spontaneous inflammation and fibrosis that mimics human NASH. Hepatology (Baltimore, Md.) |
Luther J, Khan S, Gala MK, et al. (2020) Hepatic gap junctions amplify alcohol liver injury by propagating cGAS-mediated IRF3 activation. Proceedings of the National Academy of Sciences of the United States of America |
Patel SJ, Luther J, Bohr S, et al. (2016) A Novel Resolvin-Based Strategy for Limiting Acetaminophen Hepatotoxicity. Clinical and Translational Gastroenterology. 7: e153 |
Warfield KL, Plummer EM, Sayce AC, et al. (2016) Inhibition of endoplasmic reticulum glucosidases is required for in vitro and in vivo dengue antiviral activity by the iminosugar UV-4. Antiviral Research |
Patel SJ, Milwid JM, King KR, et al. (2014) Erratum: Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure Nature Biotechnology. 32: 291-291 |
Perry ST, Buck MD, Plummer EM, et al. (2013) An iminosugar with potent inhibition of dengue virus infection in vivo. Antiviral Research. 98: 35-43 |
Patel SJ, Milwid JM, King KR, et al. (2012) Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure. Nature Biotechnology. 30: 179-83 |
Patel SJ, King KR, Bohr S, et al. (2012) 1020 Developing a Hepatoprotective Strategy for Preventing Drug-Induced Liver Toxicity and Fulminant Hepatic Failure Gastroenterology. 142: S-941 |
Patel SJ, Jindal R, King KR, et al. (2011) The inflammatory response to double stranded DNA in endothelial cells is mediated by NFκB and TNFα. Plos One. 6: e19910 |
Roach KL, King KR, Uygun BE, et al. (2009) High throughput single cell bioinformatics. Biotechnology Progress. 25: 1772-9 |