Kevin King

Affiliations: 
2001-2007 HST MIT, Harvard Medical School 
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"Kevin King"
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Publications

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Calcagno DM, Chu A, Gaul S, et al. (2022) NOD-like receptor protein 3 activation causes spontaneous inflammation and fibrosis that mimics human NASH. Hepatology (Baltimore, Md.)
Luther J, Khan S, Gala MK, et al. (2020) Hepatic gap junctions amplify alcohol liver injury by propagating cGAS-mediated IRF3 activation. Proceedings of the National Academy of Sciences of the United States of America
Patel SJ, Luther J, Bohr S, et al. (2016) A Novel Resolvin-Based Strategy for Limiting Acetaminophen Hepatotoxicity. Clinical and Translational Gastroenterology. 7: e153
Warfield KL, Plummer EM, Sayce AC, et al. (2016) Inhibition of endoplasmic reticulum glucosidases is required for in vitro and in vivo dengue antiviral activity by the iminosugar UV-4. Antiviral Research
Patel SJ, Milwid JM, King KR, et al. (2014) Erratum: Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure Nature Biotechnology. 32: 291-291
Perry ST, Buck MD, Plummer EM, et al. (2013) An iminosugar with potent inhibition of dengue virus infection in vivo. Antiviral Research. 98: 35-43
Patel SJ, Milwid JM, King KR, et al. (2012) Gap junction inhibition prevents drug-induced liver toxicity and fulminant hepatic failure. Nature Biotechnology. 30: 179-83
Patel SJ, King KR, Bohr S, et al. (2012) 1020 Developing a Hepatoprotective Strategy for Preventing Drug-Induced Liver Toxicity and Fulminant Hepatic Failure Gastroenterology. 142: S-941
Patel SJ, Jindal R, King KR, et al. (2011) The inflammatory response to double stranded DNA in endothelial cells is mediated by NFκB and TNFα. Plos One. 6: e19910
Roach KL, King KR, Uygun BE, et al. (2009) High throughput single cell bioinformatics. Biotechnology Progress. 25: 1772-9
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