C. Garrison Fathman, M.D.
Affiliations: | Stanford University, Palo Alto, CA |
Website:
https://profiles.stanford.edu/c-fathmanGoogle:
"C. Garrison Fathman"Children
Sign in to add trainee| Judith A Shizuru | grad student | 1986 | Stanford |
| Jayne S. Danska | post-doc | 1985-1991 | Stanford (Physics Tree) |
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Publications
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| Kodama K, Zhao Z, Toda K, et al. (2016) Expression-based Genome-Wide Association Study Links Vitamin D Binding Protein with Autoantigenicity in Type 1 Diabetes. Diabetes |
| Creusot RJ, Battaglia M, Roncarolo MG, et al. (2016) Concise Review: Cell-Based Therapies and Other Non-Traditional Approaches for Type 1 Diabetes. Stem Cells (Dayton, Ohio). 34: 809-19 |
| Kurnellas MP, Ghosn EE, Schartner JM, et al. (2015) Amyloid fibrils activate B-1a lymphocytes to ameliorate inflammatory brain disease. Proceedings of the National Academy of Sciences of the United States of America |
| Nair N, Mei HE, Chen SY, et al. (2015) Mass cytometry as a platform for the discovery of cellular biomarkers to guide effective rheumatic disease therapy. Arthritis Research & Therapy. 17: 127 |
| Yip L, Fuhlbrigge R, Taylor C, et al. (2015) Inflammation and hyperglycemia mediate Deaf1 splicing in the pancreatic lymph nodes via distinct pathways during type 1 diabetes. Diabetes. 64: 604-17 |
| Kurnellas MP, Schartner JM, Fathman CG, et al. (2014) Mechanisms of action of therapeutic amyloidogenic hexapeptides in amelioration of inflammatory brain disease. The Journal of Experimental Medicine. 211: 1847-56 |
| Yip L, Fathman CG. (2014) Type 1 diabetes in mice and men: gene expression profiling to investigate disease pathogenesis. Immunologic Research. 58: 340-50 |
| Creusot RJ, Giannoukakis N, Trucco M, et al. (2014) It's time to bring dendritic cell therapy to type 1 diabetes. Diabetes. 63: 20-30 |
| Yip L, Taylor C, Whiting CC, et al. (2013) Diminished adenosine A1 receptor expression in pancreatic α-cells may contribute to the pathology of type 1 diabetes. Diabetes. 62: 4208-19 |
| Yip L, Creusot RJ, Pager CT, et al. (2013) Reduced DEAF1 function during type 1 diabetes inhibits translation in lymph node stromal cells by suppressing Eif4g3. Journal of Molecular Cell Biology. 5: 99-110 |