Azucena Ramos
Affiliations: | Massachusetts Institute of Technology, Cambridge, MA, United States |
Area:
Functional genomics, acute leukemias, CAR-T therapy, chemoresistanceGoogle:
"Azucena Ramos"
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Publications
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| Liu-Lupo Y, Ham JD, Jeewajee SKA, et al. (2023) Integrated multi-omics analyses reveal homology-directed repair pathway as a unique dependency in near-haploid leukemia. Blood Cancer Journal. 13: 92 |
| Gordon KS, Kyung T, Perez CR, et al. (2023) Author Correction: Screening for CD19-specific chimaeric antigen receptors with enhanced signalling via a barcoded library of intracellular domains. Nature Biomedical Engineering |
| Gordon KS, Kyung T, Perez CR, et al. (2022) Screening for CD19-specific chimaeric antigen receptors with enhanced signalling via a barcoded library of intracellular domains. Nature Biomedical Engineering |
| Wojtaszek JL, Chatterjee N, Najeeb J, et al. (2019) A Small Molecule Targeting Mutagenic Translesion Synthesis Improves Chemotherapy. Cell |
| Ramos A, Barea LRM, Puissant A, et al. (2018) Abstract 2358: Uncovering novel mechanisms of resistance in AML using integrative functional genomics Cancer Research. 78: 2358-2358 |
| Ramos A, Hemann MT. (2017) Drugs, Bugs, and Cancer: Fusobacterium nucleatum Promotes Chemoresistance in Colorectal Cancer. Cell. 170: 411-413 |
| Fenouille N, Bassil CF, Ben-Sahra I, et al. (2017) The creatine kinase pathway is a metabolic vulnerability in EVI1-positive acute myeloid leukemia. Nature Medicine |
| Pikman Y, Puissant A, Alexe G, et al. (2016) Targeting MTHFD2 in acute myeloid leukemia. The Journal of Experimental Medicine |
| Lindblad O, Cordero E, Puissant A, et al. (2016) Aberrant activation of the PI3K/mTOR pathway promotes resistance to sorafenib in AML. Oncogene |
| Puissant A, Fenouille N, Bassil CF, et al. (2016) Targeting the Creatine Kinase Pathway in EVI1-Positive Acute Myeloid Leukemia Blood. 128: 523-523 |