| Year |
Citation |
Score |
| 2024 |
Chen JL, Kuo CC. Inhibition of resurgent Na currents by rufinamide. Neuropharmacology. 109835. PMID 38228283 DOI: 10.1016/j.neuropharm.2024.109835 |
0.327 |
|
| 2020 |
Tai CH, Pan MK, Tseng SH, Wang TR, Kuo CC. Author Correction: Hyperpolarization of the subthalamic nucleus alleviates hyperkinetic movement disorders. Scientific Reports. 10: 9840. PMID 32528132 DOI: 10.1038/S41598-020-67067-6 |
0.643 |
|
| 2020 |
Tai CH, Pan MK, Tseng SH, Wang TR, Kuo CC. Hyperpolarization of the subthalamic nucleus alleviates hyperkinetic movement disorders. Scientific Reports. 10: 8278. PMID 32427942 DOI: 10.1038/S41598-020-65211-W |
0.693 |
|
| 2019 |
Chen YS, Tu YC, Lai YC, Liu E, Yang YC, Kuo CC. Desensitization of NMDA channels requires ligand binding to both GluN1 and GluN2 subunits to constrict the pore beside the activation gate. Journal of Neurochemistry. e14939. PMID 31821563 DOI: 10.1111/Jnc.14939 |
0.382 |
|
| 2019 |
Huang CW, Lai HJ, Huang PY, Lee MJ, Kuo CC. Anomalous enhancement of resurgent Na currents at high temperatures by SCN9A mutations underlies the episodic heat-enhanced pain in inherited erythromelalgia. Scientific Reports. 9: 12251. PMID 31439884 DOI: 10.1038/S41598-019-48672-6 |
0.356 |
|
| 2019 |
Lo YC, Kuo CC. Temperature dependence of the biophysical mechanisms underlying the apt inhibition and enhancement effect of amiodarone on hERG channels. Molecular Pharmacology. PMID 31253645 DOI: 10.1124/Mol.119.116400 |
0.421 |
|
| 2016 |
Tu YC, Yang YC, Kuo CC. Modulation of NMDA channel gating by Ca(2+) and Cd(2+) binding to the external pore mouth. Scientific Reports. 6: 37029. PMID 27848984 DOI: 10.1038/Srep37029 |
0.361 |
|
| 2016 |
Pan MK, Kuo SH, Tai CH, Liou JY, Pei JC, Chang CY, Wang YM, Liu WC, Wang TR, Lai WS, Kuo CC. Neuronal firing patterns outweigh circuitry oscillations in parkinsonian motor control. The Journal of Clinical Investigation. PMID 27797341 DOI: 10.1172/Jci88170 |
0.679 |
|
| 2016 |
Huang CW, Lai HJ, Huang PY, Lee MJ, Kuo CC. The Biophysical Basis Underlying Gating Changes in the p.V1316A Mutant Nav1.7 Channel and the Molecular Pathogenesis of Inherited Erythromelalgia. Plos Biology. 14: e1002561. PMID 27653502 DOI: 10.1371/Journal.Pbio.1002561 |
0.386 |
|
| 2016 |
Huang CW, Kuo CC. A synergistic blocking effect of Mg(2+) and spermine on the inward rectifier K(+) (Kir2.1) channel pore. Scientific Reports. 6: 21493. PMID 26869275 DOI: 10.1038/Srep21493 |
0.386 |
|
| 2015 |
Hsieh CP, Kuo CC, Huang CW. Driving force-dependent block by internal Ba(2+) on the Kir2.1 channel: Mechanistic insight into inward rectification. Biophysical Chemistry. 202: 40-57. PMID 25913355 DOI: 10.1016/J.Bpc.2015.04.003 |
0.396 |
|
| 2015 |
Huang CW, Kuo CC. Flow- and voltage-dependent blocking effect of ethosuximide on the inward rectifier K(+) (Kir2.1) channel. PflüGers Archiv : European Journal of Physiology. 467: 1733-46. PMID 25220134 DOI: 10.1007/S00424-014-1611-2 |
0.439 |
|
| 2014 |
Tu YC, Kuo CC. The differential contribution of GluN1 and GluN2 to the gating operation of the NMDA receptor channel. Pflugers Archiv : European Journal of Physiology. PMID 25339225 DOI: 10.1007/S00424-014-1630-Z |
0.344 |
|
| 2014 |
Pan MK, Tai CH, Liu WC, Pei JC, Lai WS, Kuo CC. Deranged NMDAergic cortico-subthalamic transmission underlies parkinsonian motor deficits. The Journal of Clinical Investigation. 124: 4629-41. PMID 25202982 DOI: 10.1172/Jci75587 |
0.694 |
|
| 2014 |
Huang CW, Kuo CC. Gating of the kir2.1 channel at the bundle crossing region by intracellular spermine and other cations. Journal of Cellular Physiology. 229: 1703-21. PMID 24633623 DOI: 10.1002/Jcp.24616 |
0.39 |
|
| 2014 |
Yang YC, Tai CH, Pan MK, Kuo CC. The T-type calcium channel as a new therapeutic target for Parkinson's disease. Pflã¼Gers Archiv : European Journal of Physiology. 466: 747-55. PMID 24531801 DOI: 10.1007/S00424-014-1466-6 |
0.736 |
|
| 2014 |
Huang CW, Kuo CC. The bundle crossing region is responsible for the inwardly rectifying internal spermine block of the Kir2.1 channel. Pflã¼Gers Archiv : European Journal of Physiology. 466: 275-93. PMID 23873351 DOI: 10.1007/S00424-013-1322-0 |
0.369 |
|
| 2012 |
Tai CH, Pan MK, Lin JJ, Huang CS, Yang YC, Kuo CC. Subthalamic discharges as a causal determinant of parkinsonian motor deficits. Annals of Neurology. 72: 464-76. PMID 23034918 DOI: 10.1002/Ana.23618 |
0.723 |
|
| 2011 |
Yang YC, Lin S, Chang PC, Lin HC, Kuo CC. Functional extension of amino acid triads from the fourth transmembrane segment (S4) into its external linker in Shaker K(+) channels. The Journal of Biological Chemistry. 286: 37503-14. PMID 21900243 DOI: 10.1074/Jbc.M111.237792 |
0.32 |
|
| 2011 |
Tai CH, Yang YC, Pan MK, Huang CS, Kuo CC. Modulation of subthalamic T-type Ca(2+) channels remedies locomotor deficits in a rat model of Parkinson disease. The Journal of Clinical Investigation. 121: 3289-305. PMID 21737877 DOI: 10.1172/Jci46482 |
0.723 |
|
| 2011 |
Chen CP, Ganguly A, Lu CY, Chen TY, Kuo CC, Chen RS, Tu WH, Fischer WB, Chen KH, Chen LC. Ultrasensitive in situ label-free DNA detection using a GaN nanowire-based extended-gate field-effect-transistor sensor. Analytical Chemistry. 83: 1938-43. PMID 21351780 DOI: 10.1021/Ac102489Y |
0.355 |
|
| 2010 |
Pan MK, Tai CH, Kuo CC. [Parkinson's disease and cortico-Basal Ganglia circuits]. Acta Neurologica Taiwanica. 19: 213-22. PMID 20824544 |
0.666 |
|
| 2010 |
Yang YC, Huang CS, Kuo CC. Lidocaine, carbamazepine, and imipramine have partially overlapping binding sites and additive inhibitory effect on neuronal Na+ channels. Anesthesiology. 113: 160-74. PMID 20526191 DOI: 10.1097/Aln.0B013E3181Dc1Dd6 |
0.38 |
|
| 2010 |
Yang YC, Lee CH, Kuo CC. Ionic flow enhances low-affinity binding: a revised mechanistic view into Mg2+ block of NMDA receptors. The Journal of Physiology. 588: 633-50. PMID 20026615 DOI: 10.1113/Jphysiol.2009.178913 |
0.386 |
|
| 2009 |
Yang YC, Hsieh JY, Kuo CC. The external pore loop interacts with S6 and S3-S4 linker in domain 4 to assume an essential role in gating control and anticonvulsant action in the Na(+) channel. The Journal of General Physiology. 134: 95-113. PMID 19635852 DOI: 10.1085/Jgp.200810158 |
0.39 |
|
| 2008 |
Chang HR, Kuo CC. The activation gate and gating mechanism of the NMDA receptor. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 28: 1546-56. PMID 18272676 DOI: 10.1523/Jneurosci.3485-07.2008 |
0.323 |
|
| 2007 |
Chang HR, Kuo CC. Extracellular proton-modulated pore-blocking effect of the anticonvulsant felbamate on NMDA channels. Biophysical Journal. 93: 1981-92. PMID 17513365 DOI: 10.1529/Biophysj.106.103176 |
0.375 |
|
| 2007 |
Chang HR, Kuo CC. Characterization of the gating conformational changes in the felbamate binding site in NMDA channels. Biophysical Journal. 93: 456-66. PMID 17468173 DOI: 10.1529/Biophysj.106.098095 |
0.364 |
|
| 2007 |
Yang YC, Own CJ, Kuo CC. A hydrophobic element secures S4 voltage sensor in position in resting Shaker K+ channels. The Journal of Physiology. 582: 1059-72. PMID 17412765 DOI: 10.1113/Jphysiol.2007.131490 |
0.403 |
|
| 2005 |
Yang YC, Kuo CC. An inactivation stabilizer of the Na+ channel acts as an opportunistic pore blocker modulated by external Na+. The Journal of General Physiology. 125: 465-81. PMID 15824190 DOI: 10.1085/Jgp.200409156 |
0.408 |
|
| 2004 |
Kuo CC, Chen WY, Yang YC. Block of tetrodotoxin-resistant Na+ channel pore by multivalent cations: gating modification and Na+ flow dependence. The Journal of General Physiology. 124: 27-42. PMID 15226363 DOI: 10.1085/Jgp.200409054 |
0.413 |
|
| 2004 |
Kuo CC, Lin BJ, Chang HR, Hsieh CP. Use-dependent inhibition of the N-methyl-D-aspartate currents by felbamate: a gating modifier with selective binding to the desensitized channels. Molecular Pharmacology. 65: 370-80. PMID 14742679 DOI: 10.1124/Mol.65.2.370 |
0.346 |
|
| 2003 |
Yang YC, Kuo CC. The position of the fourth segment of domain 4 determines status of the inactivation gate in Na+ channels. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 23: 4922-30. PMID 12832514 DOI: 10.1523/Jneurosci.23-12-04922.2003 |
0.373 |
|
| 2002 |
Yang YC, Kuo CC. Inhibition of Na(+) current by imipramine and related compounds: different binding kinetics as an inactivation stabilizer and as an open channel blocker. Molecular Pharmacology. 62: 1228-37. PMID 12391287 DOI: 10.1124/Mol.62.5.1228 |
0.419 |
|
| 2002 |
Kuo CC, Lin TJ, Hsieh CP. Effect of Na(+) flow on Cd(2+) block of tetrodotoxin-resistant Na(+) channels. The Journal of General Physiology. 120: 159-72. PMID 12149278 DOI: 10.1085/Jgp.20018536 |
0.407 |
|
| 2001 |
Kuo CC, Yang S. Recovery from inactivation of T-type Ca2+ channels in rat thalamic neurons Journal of Neuroscience. 21: 1884-1892. PMID 11245673 DOI: 10.1523/Jneurosci.21-06-01884.2001 |
0.433 |
|
| 2000 |
Kuo CC, Liao SY. Facilitation of recovery from inactivation by external NA+ and location of the activation gate in neuronal NA+ channels Journal of Neuroscience. 20: 5639-5646. PMID 10908601 DOI: 10.1523/Jneurosci.20-15-05639.2000 |
0.407 |
|
| 1999 |
Kuo CC, Chen FP. Zn2+ modulation of neuronal transient K+ current: Fast and selective binding to the deactivated channels Biophysical Journal. 77: 2552-2562. PMID 10545356 DOI: 10.1016/S0006-3495(99)77090-6 |
0.385 |
|
| 1999 |
Shieh RC, Chang JC, Kuo CC. K + binding sites and interactions between permeating K+ ions at the external pore mouth of an inward rectifier K+ channel (Kir2.1) Journal of Biological Chemistry. 274: 17424-17430. PMID 10364171 DOI: 10.1074/Jbc.274.25.17424 |
0.365 |
|
| 1998 |
Kuo CC. Imipramine inhibition of transient K+ current: An external open channel blocker preventing fast inactivation Biophysical Journal. 75: 2845-2857. PMID 9826606 DOI: 10.1016/S0006-3495(98)77727-6 |
0.441 |
|
| 1997 |
Kuo CC, Lu L. Characterization of lamotrigine inhibition of Na+ channels in rat hippocampal neurones British Journal of Pharmacology. 121: 1231-1238. PMID 9249262 DOI: 10.1038/Sj.Bjp.0701221 |
0.422 |
|
| 1997 |
Kuo CC, Chen RS, Lu L, Chen RC. Carbamazepine inhibition of neuronal Na+ currents: quantitative distinction from phenytoin and possible therapeutic implications. Molecular Pharmacology. 51: 1077-83. PMID 9187275 DOI: 10.1124/Mol.51.6.1077 |
0.55 |
|
| 1997 |
Kuo CC. Deactivation retards recovery from inactivation in Shaker K+ channels Journal of Neuroscience. 17: 3436-3444. PMID 9133369 DOI: 10.1523/Jneurosci.17-10-03436.1997 |
0.383 |
|
| 1994 |
Kuo CC, Bean BP. Na+ channels must deactivate to recover from inactivation. Neuron. 12: 819-29. PMID 8161454 DOI: 10.1016/0896-6273(94)90335-2 |
0.64 |
|
| 1994 |
Kuo CC, Bean BP. Slow binding of phenytoin to inactivated sodium channels in rat hippocampal neurons. Molecular Pharmacology. 46: 716-25. PMID 7969051 |
0.606 |
|
| 1993 |
Kuo CC, Hess P. Block of the L-type Ca2+ channel pore by external and internal Mg2+ in rat phaeochromocytoma cells. The Journal of Physiology. 466: 683-706. PMID 8410712 DOI: 10.1113/Jphysiol.1993.Sp019740 |
0.535 |
|
| 1993 |
Kuo CC, Hess P. Characterization of the high-affinity Ca2+ binding sites in the L-type Ca2+ channel pore in rat phaeochromocytoma cells. The Journal of Physiology. 466: 657-82. PMID 8410711 DOI: 10.1113/Jphysiol.1993.Sp019739 |
0.538 |
|
| 1993 |
Kuo CC, Hess P. Ion permeation through the L-type Ca2+ channel in rat phaeochromocytoma cells: two sets of ion binding sites in the pore. The Journal of Physiology. 466: 629-55. PMID 8410710 DOI: 10.1113/Jphysiol.1993.Sp019738 |
0.546 |
|
| 1993 |
Kuo CC, Bean BP. G-protein modulation of ion permeation through N-type calcium channels. Nature. 365: 258-62. PMID 8396731 DOI: 10.1038/365258A0 |
0.621 |
|
| 1992 |
Kuo CC, Hess P. A functional view of the entrances of L-type Ca2+ channels: estimates of the size and surface potential at the pore mouths. Neuron. 9: 515-26. PMID 1381926 DOI: 10.1016/0896-6273(92)90189-K |
0.55 |
|
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