Philine A. Wangemann
Affiliations: | Department of Anatomy and Physiology | Kansas State University, Manhattan, KS, United States |
Area:
Animal Physiology Biology, Molecular BiologyGoogle:
"Philine Wangemann"Children
Sign in to add traineeRuchira Singh | grad student | 2008 | Kansas State University |
Xiangming Li | grad student | 2013 | Kansas State University |
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Publications
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Jeong J, Kim JY, Hong H, et al. (2019) P2RX2 and P2RX4 receptors mediate cation absorption in transitional cells and supporting cells of the utricular macula. Hearing Research. 386: 107860 |
Kim MA, Kim SH, Ryu N, et al. (2019) Gene therapy for hereditary hearing loss by SLC26A4 mutations in mice reveals distinct functional roles of pendrin in normal hearing. Theranostics. 9: 7184-7199 |
Nakanishi H, Kurima K, Pan B, et al. (2018) Tmc2 expression partially restores auditory function in a mouse model of DFNB7/B11 deafness caused by loss of Tmc1 function. Scientific Reports. 8: 12125 |
Kudo T, Wangemann P, Marcus DC. (2018) Claudin expression during early postnatal development of the murine cochlea Bmc Physiology. 18: 1-1 |
Honda K, Kim SH, Kelly MC, et al. (2017) Molecular architecture underlying fluid absorption by the developing inner ear. Elife. 6 |
Honda K, Kim SH, Kelly MC, et al. (2017) Author response: Molecular architecture underlying fluid absorption by the developing inner ear Elife |
Miyazaki H, Wangemann P, Marcus DC. (2016) The gastric H,K-ATPase in stria vascularis contributes to pH regulation of cochlear endolymph but not to K secretion. Bmc Physiology. 17: 1 |
Nishio A, Ito T, Cheng H, et al. (2016) Slc26a4 Expression Prevents Fluctuation of Hearing in a Mouse Model of Large Vestibular Aqueduct Syndrome. Neuroscience |
Ito T, Nishio A, Wangemann P, et al. (2015) Progressive irreversible hearing loss is caused by stria vascularis degeneration in an Slc26a4-insufficient mouse model of large vestibular aqueduct syndrome. Neuroscience. 310: 188-97 |
Lu J, Aguilar A, Zou B, et al. (2015) Chemical synthesis of tetracyclic terpenes and evaluation of antagonistic activity on endothelin-A receptors and voltage-gated calcium channels. Bioorganic & Medicinal Chemistry. 23: 5985-98 |