Andrea E. Knowlton, Ph.D.
Affiliations: | 2012 | University of Florida, Gainesville, Gainesville, FL, United States |
Area:
Microbiology Biology, Cell Biology, General BiologyGoogle:
"Andrea Knowlton"Parents
Sign in to add mentor| Scott Grieshaber | grad student | 2012 | UF Gainesville | |
| (Infection with Chlamydia trachomatis results in detrimental host cell defects dependent upon cell cycle progression.) | ||||
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Publications
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| Pham K, Delitto D, Knowlton AE, et al. (2016) Isolation of Pancreatic Cancer Cells from a Patient-Derived Xenograft Model Allows for Practical Expansion and Preserved Heterogeneity in Culture. The American Journal of Pathology |
| Delitto D, Perez C, Han S, et al. (2015) Downstream mediators of the intratumoral interferon response suppress antitumor immunity, induce gemcitabine resistance and associate with poor survival in human pancreatic cancer. Cancer Immunology, Immunotherapy : Cii |
| Brown HM, Knowlton AE, Snavely E, et al. (2014) Multinucleation during C. trachomatis infections is caused by the contribution of two effector pathways. Plos One. 9: e100763 |
| Richards TS, Knowlton AE, Grieshaber SS. (2013) Chlamydia trachomatis homotypic inclusion fusion is promoted by host microtubule trafficking. Bmc Microbiology. 13: 185 |
| Knowlton AE, Fowler LJ, Patel RK, et al. (2013) Chlamydia induces anchorage independence in 3T3 cells and detrimental cytological defects in an infection model. Plos One. 8: e54022 |
| Brown HM, Knowlton AE, Grieshaber SS. (2012) Chlamydial infection induces host cytokinesis failure at abscission. Cellular Microbiology. 14: 1554-67 |
| Knowlton AE, Brown HM, Richards TS, et al. (2011) Chlamydia trachomatis infection causes mitotic spindle pole defects independently from its effects on centrosome amplification. Traffic (Copenhagen, Denmark). 12: 854-66 |