Adam G. Goodwill

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2011-2015  
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"Adam G. Goodwill"
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Tune JD, Goodwill AG, Baker HE, et al. (2022) Chronic high-rate pacing induces heart failure with preserved ejection fraction-like phenotype in Ossabaw swine. Basic Research in Cardiology. 117: 50
Baker HE, Tune JD, Mather KJ, et al. (2022) Acute SGLT-2i treatment improves cardiac efficiency during myocardial ischemia independent of Na+/H+ exchanger-1. International Journal of Cardiology
Goodwill AG, Baker HE, Dick GM, et al. (2021) Mineralocorticoid receptor blockade normalizes coronary resistance in obese swine independent of functional alterations in K channels. Basic Research in Cardiology. 116: 35
Tune JD, Baker HE, Berwick ZC, et al. (2020) Distinct hemodynamic responses to (Pyr)Apelin-13 in large animal models. American Journal of Physiology. Heart and Circulatory Physiology
Baker H, Tune J, Mather K, et al. (2020) Inhibition of Sodium Glucose Cotransporter‐2 Improves Cardiac Efficiency During Regional Myocardial Ischemia Independent of Sodium/Hydrogen Exchanger‐1 The Faseb Journal. 34: 1-1
Tune JD, Goodwill AG, Kiel A, et al. (2019) Disentangling the Gordian knot of local metabolic control of coronary blood flow. American Journal of Physiology. Heart and Circulatory Physiology
Baker HE, Tune JD, Goodwill AG, et al. (2019) Benefits of Empagliflozin Beyond Enhancing Myocardial Energetics? Journal of the American College of Cardiology. 74: 825-826
Baker HE, Kiel AM, Luebbe ST, et al. (2019) Inhibition of sodium-glucose cotransporter-2 preserves cardiac function during regional myocardial ischemia independent of alterations in myocardial substrate utilization. Basic Research in Cardiology. 114: 25
Clark HE, Baker HE, Goodwill AG, et al. (2019) Obesity decreases the contribution of Kv channels to hypoxic coronary vasodilation Proceedings of Imprs. 2
Blaettner BS, Baker HE, Goodwill AG, et al. (2019) Inhibition of Type 2 Sodium-Glucose Transporters and Na+/H+ Exchanger-1 produces similar cardioprotective effects in response to ischemiareperfusion injury Proceedings of Imprs. 2
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