Thomas J. Baranski

Biology & Biomedical Sciences (Molecular Cell Biology) Washington University, Saint Louis, St. Louis, MO 
Cell Biology, Molecular Biology
"Thomas Baranski"
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Musselman LP, Fink JL, Baranski TJ. (2016) CoA protects against the deleterious effects of caloric overload in Drosophila. Journal of Lipid Research
Hirabayashi S, Baranski TJ, Cagan RL. (2013) Transformed Drosophila cells evade diet-mediated insulin resistance through wingless signaling. Cell. 154: 664-75
Buescher JL, Musselman LP, Wilson CA, et al. (2013) Evidence for transgenerational metabolic programming in Drosophila. Disease Models & Mechanisms. 6: 1123-32
Nichols AS, Floyd DH, Bruinsma SP, et al. (2013) Frizzled receptors signal through G proteins. Cellular Signalling. 25: 1468-75
Pendse J, Ramachandran PV, Na J, et al. (2013) A Drosophila functional evaluation of candidates from human genome-wide association studies of type 2 diabetes and related metabolic traits identifies tissue-specific roles for dHHEX. Bmc Genomics. 14: 136
Musselman LP, Fink JL, Ramachandran PV, et al. (2013) Role of fat body lipogenesis in protection against the effects of caloric overload in Drosophila. The Journal of Biological Chemistry. 288: 8028-42
Na J, Musselman LP, Pendse J, et al. (2013) A Drosophila model of high sugar diet-induced cardiomyopathy. Plos Genetics. 9: e1003175
Nikiforovich GV, Baranski TJ. (2012) Structural mechanisms of constitutive activation in the C5a receptors with mutations in the extracellular loops: molecular modeling study. Proteins. 80: 71-80
Nikiforovich GV, Taylor CM, Marshall GR, et al. (2011) Difference between restoring and predicting 3D structures of the loops in G-protein-coupled receptors by molecular modeling. Proceedings of the National Academy of Sciences of the United States of America. 108: E341; author reply E
Musselman LP, Fink JL, Narzinski K, et al. (2011) A high-sugar diet produces obesity and insulin resistance in wild-type Drosophila. Disease Models & Mechanisms. 4: 842-9
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