Michael Kastan
Affiliations: | Johns Hopkins University, Baltimore, MD |
Area:
Cell Biology, OncologyGoogle:
"Michael Kastan"
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Publications
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| McCann JJ, Fleenor DE, Chen J, et al. (2023) Participation of ATM, SMG1, and DDX5 in a DNA Damage-induced Alternative Splicing Pathway. Radiation Research |
| Bass TE, Fleenor DE, Burrell PE, et al. (2022) ATM Regulation of the Cohesin Complex is Required for Repression of DNA Replication and Transcription in the Vicinity of DNA Double Strand Breaks. Molecular Cancer Research : McR |
| Chen J, Zhang D, Qin X, et al. (2021) DNA-Damage-Induced Alternative Splicing of p53. Cancers. 13 |
| Chen J, Kastan MB. (2017) A novel DNA damage-induced alternative splicing pathway that regulates p53 and cellular senescence markers. Oncoscience. 4: 122-123 |
| Liu X, Li F, Huang Q, et al. (2017) Self-inflicted DNA double-strand breaks sustain tumorigenicity and stemness of cancer cells. Cell Research |
| Chen J, Crutchley J, Zhang D, et al. (2017) Identification of a DNA Damage-induced Alternative Splicing Pathway that Regulates p53 and Cellular Senescence Markers. Cancer Discovery |
| Kastan MB. (2017) Commentary on "Participation of p53 Protein in the Cellular Response to DNA Damage". Cancer Research. 76: 3663-5 |
| Bakkenist CJ, Kastan MB. (2015) Chromatin perturbations during the DNA damage response in higher eukaryotes. Dna Repair. 36: 8-12 |
| Goldstein M, Kastan MB. (2015) Repair versus Checkpoint Functions of BRCA1 Are Differentially Regulated by Site of Chromatin Binding. Cancer Research. 75: 2699-707 |
| Henriksson S, Rassoolzadeh H, Hedström E, et al. (2014) The scaffold protein WRAP53β orchestrates the ubiquitin response critical for DNA double-strand break repair. Genes & Development. 28: 2726-38 |