David E. Cochrane
Affiliations: | Tufts University, Boston |
Area:
Cell Biology, Animal Physiology Biology, PathologyGoogle:
"David Cochrane"
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Publications
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| Carraway RE, Cochrane DE. (2012) Enhanced vascular permeability is hypothesized to promote inflammation-induced carcinogenesis and tumor development via extravasation of large molecular proteins into the tissue. Medical Hypotheses. 78: 738-43 |
| Cochrane DE, Carraway RE, Harrington K, et al. (2011) HMC-1 human mast cells synthesize neurotensin (NT) precursor, secrete bioactive NT-like peptide(s) and express NT receptor NTS1. Inflammation Research : Official Journal of the European Histamine Research Society ... [Et Al.]. 60: 1139-51 |
| Carraway RE, Hassan S, Cochrane DE. (2006) Regulation of neurotensin receptor function by the arachidonic acid-lipoxygenase pathway in prostate cancer PC3 cells. Prostaglandins, Leukotrienes, and Essential Fatty Acids. 74: 93-107 |
| Scarpa RC, Carraway RE, Cochrane DE. (2005) Insulin-like growth factor (IGF) induced proliferation of human lung fibroblasts is enhanced by neurotensin. Peptides. 26: 2201-10 |
| Scarpa RC, Carraway RE, Cochrane DE. (2004) The effect of neurotensin on insulin-induced proliferation of human fibroblasts. Peptides. 25: 1159-69 |
| Carraway RE, Hassan S, Cochrane DE. (2004) Polyphenolic antioxidants mimic the effects of 1,4-dihydropyridines on neurotensin receptor function in PC3 cells. The Journal of Pharmacology and Experimental Therapeutics. 309: 92-101 |
| Theoharides TC, Cochrane DE. (2004) Critical role of mast cells in inflammatory diseases and the effect of acute stress. Journal of Neuroimmunology. 146: 1-12 |
| Carraway RE, Gui X, Cochrane DE. (2003) Ca2+ channel blockers enhance neurotensin (NT) binding and inhibit NT-induced inositol phosphate formation in prostate cancer PC3 cells. The Journal of Pharmacology and Experimental Therapeutics. 307: 640-50 |
| Cochrane DE, Carraway RE, Miller LA, et al. (2003) Histamine releasing peptide (HRP) has proinflammatory effects and is present at sites of inflammation. Biochemical Pharmacology. 66: 331-42 |
| Carraway RE, Mitra SP, Cochrane DE. (2001) Pro-xenopsin(s) in vesicles of mammalian brain, liver, stomach and intestine is apparently released into blood and cerebral spinal fluid. Regulatory Peptides. 95: 115-24 |