Glenn I. Fishman
Affiliations: | Icahn School of Medicine at Mount Sinai, New York, NY, United States |
Area:
Molecular Biology, Human Development, Animal Physiology BiologyGoogle:
"Glenn Fishman"Children
Sign in to add trainee| Stacey L. Rentschler | grad student | 2002 | Mount Sinai School of Medicine |
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Publications
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| Sun B, Ni M, Li Y, et al. (2024) Inositol 1,4,5-Trisphosphate Receptor 1 Gain-of-Function Increases the Risk for Cardiac Arrhythmias in Mice and Humans. Circulation |
| Kim EE, Shekhar A, Ramachandran J, et al. (2023) The transcription factor EBF1 non-cell-autonomously regulates cardiac growth and differentiation. Development (Cambridge, England). 150 |
| Barefield DY, Yamakawa S, Tahtah I, et al. (2022) Partial and complete loss of myosin binding protein H-like cause cardiac conduction defects. Journal of Molecular and Cellular Cardiology. 169: 28-40 |
| Blackwell DJ, Faggioni M, Wleklinski MJ, et al. (2022) The Purkinje-myocardial junction is the anatomic origin of ventricular arrhythmia in CPVT. Jci Insight. 7 |
| Delgado C, Bu L, Zhang J, et al. (2021) Neural cell adhesion molecule is required for ventricular conduction system development. Development (Cambridge, England). 148 |
| Delgado C, Bu L, Zhang J, et al. (2021) Neural cell adhesion molecule (NCAM-1) is required for ventricular conduction system development. Development (Cambridge, England) |
| Park DS, Shekhar A, Santucci Iii J, et al. (2020) Ionic Mechanisms of Impulse Propagation Failure in the FHF2-Deficient Heart. Circulation Research |
| Shekhar A, Aristizabal O, Fishman GI, et al. (2020) Characterization of vortex flow in a mouse model of ventricular dyssynchrony by plane-wave ultrasound using hexplex processing. Ieee Transactions On Ultrasonics, Ferroelectrics, and Frequency Control |
| Fishman GI. (2020) TRANSCRIPTIONAL REGULATION OF THE CARDIAC CONDUCTION SYSTEM. Transactions of the American Clinical and Climatological Association. 131: 48-54 |
| Himelman E, Lillo MA, Nouet J, et al. (2020) Prevention of Connexin43 remodeling protects against duchenne muscular dystrophy cardiomyopathy. The Journal of Clinical Investigation |