Nazumi A. Yamada, Ph.D.
Affiliations: | 2002 | University of North Carolina, Chapel Hill, Chapel Hill, NC |
Area:
Molecular BiologyGoogle:
"Nazumi Yamada"Parents
Sign in to add mentor| Rosann A. Farber | grad student | 2002 | UNC Chapel Hill | |
| (Microsatellite instability in the absence of mismatch repair defects.) | ||||
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Publications
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| Elso C, Lu X, Morrison S, et al. (2008) Germline translocations in mice: unique tools for analyzing gene function and long-distance regulatory mechanisms. Journal of the National Cancer Institute. Monographs. 91-5 |
| Hinz JM, Nham PB, Yamada NA, et al. (2006) Four human FANCG polymorphic variants show normal biological function in hamster CHO cells. Mutation Research. 602: 34-42 |
| Hinz JM, Yamada NA, Salazar EP, et al. (2005) Influence of double-strand-break repair pathways on radiosensitivity throughout the cell cycle in CHO cells. Dna Repair. 4: 782-92 |
| Miller KA, Hinz JM, Yamada NA, et al. (2005) Nuclear localization of Rad51B is independent of Rad51C and BRCA2. Mutagenesis. 20: 57-63 |
| Thompson LH, Hinz JM, Yamada NA, et al. (2005) How Fanconi anemia proteins promote the four Rs: Replication, recombination, repair, and recovery Environmental and Molecular Mutagenesis. 45: 128-142 |
| Tebbs RS, Hinz JM, Yamada NA, et al. (2005) New insights into the Fanconi anemia pathway from an isogenic FancG hamster CHO mutant. Dna Repair. 4: 11-22 |
| Lamerdin JE, Yamada NA, George JW, et al. (2004) Characterization of the hamster FancG/Xrcc9 gene and mutations in CHO UV40 and NM3 Mutagenesis. 19: 237-244 |
| Yamada NA, Hinz JM, Kopf VL, et al. (2004) XRCC3 ATPase activity is required for normal XRCC3-Rad51C complex dynamics and homologous recombination. The Journal of Biological Chemistry. 279: 23250-4 |
| Yamada NA, Parker JM, Farber RA. (2003) Mutation frequency analysis of mononucleotide and dinucleotide repeats after oxidative stress. Environmental and Molecular Mutagenesis. 42: 75-84 |
| Yamada NA, Castro A, Farber RA. (2003) Variation in the extent of microsatellite instability in human cell lines with defects in different mismatch repair genes. Mutagenesis. 18: 277-82 |