Mong-Hong Lee
Affiliations: | The University of Texas Graduate School of Biomedical Sciences at Houston |
Area:
Molecular Biology, Genetics, Cell BiologyGoogle:
"Mong-Hong Lee"Parents
Sign in to add mentor| Graham F. Hatfull | grad student | 1988-1993 | University of Pittsburgh (Chemistry Tree) |
Children
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Publications
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| Jin H, Huang X, Pan Q, et al. (2024) The EIF3H-HAX1 axis increases RAF-MEK-ERK signaling activity to promote colorectal cancer progression. Nature Communications. 15: 2551 |
| Li K, Zhang J, Lyu H, et al. (2024) CSN6-SPOP-HMGCS1 Axis Promotes Hepatocellular Carcinoma Progression via YAP1 Activation. Advanced Science (Weinheim, Baden-Wurttemberg, Germany). e2306827 |
| Pan Q, Yu F, Jin H, et al. (2023) eIF3f Mediates SGOC Pathway Reprogramming by Enhancing Deubiquitinating Activity in Colorectal Cancer. Advanced Science (Weinheim, Baden-Wurttemberg, Germany). e2300759 |
| Yang Z, Zou S, Zhang Y, et al. (2023) ACTL6A protects gastric cancer cells against ferroptosis through induction of glutathione synthesis. Nature Communications. 14: 4193 |
| Wu W, Zhao Y, Qin B, et al. (2023) Non-canonical role of UCKL1 on ferroptosis defence in colorectal cancer. Ebiomedicine. 93: 104650 |
| Wei W, Qin B, Wen W, et al. (2023) FBXW7β loss-of-function enhances FASN-mediated lipogenesis and promotes colorectal cancer growth. Signal Transduction and Targeted Therapy. 8: 187 |
| Xu H, Luo H, Zhang J, et al. (2023) Therapeutic potential of anticancer effects in colorectal cancer. Gut Microbes. 15: 2186114 |
| Zou S, Qin B, Yang Z, et al. (2022) CSN6 mediates nucleotide metabolism to promote tumor development and chemoresistance in colorectal cancer. Cancer Research |
| Zhang B, Liu Q, Wen W, et al. (2022) The chromatin remodeler CHD6 promotes colorectal cancer development by regulating TMEM65-mediated mitochondrial dynamics via EGF and Wnt signaling. Cell Discovery. 8: 130 |
| Meng X, Peng J, Xie X, et al. (2022) Roles of lncRNA LVBU in regulating urea cycle/polyamine synthesis axis to promote colorectal carcinoma progression. Oncogene |