William E. Lento, Ph.D.
Affiliations: | 2010 | Molecular Cancer Biology | Duke University, Durham, NC |
Area:
Cell Biology, Molecular BiologyGoogle:
"William Lento"Parents
Sign in to add mentor| Tannishtha Reya | grad student | 2010 | Duke | |
| (Activation of developmental signaling pathways in hematopoietic stem cell regeneration.) | ||||
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Publications
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| Racioppi L, Nelson ER, Huang W, et al. (2019) CaMKK2 in myeloid cells is a key regulator of the immune-suppressive microenvironment in breast cancer. Nature Communications. 10: 2450 |
| Klamer SE, Dorland YL, Kleijer M, et al. (2018) TGFBI expressed by bone marrow niche cells and hematopoietic stem and progenitor cells regulates hematopoiesis. Stem Cells and Development |
| Racioppi L, Lento W, Huang W, et al. (2017) Calcium/calmodulin-dependent kinase kinase 2 regulates hematopoietic stem and progenitor cell regeneration. Cell Death & Disease. 8: e3076 |
| Adamia S, Nemeth J, Bhatt S, et al. (2016) FLT3 Splice Variant (FLT3Va) As a Potential Immunotherapeutic Target in Patients with Acute Myeloid Leukemia (AML) Blood. 128: 1681-1681 |
| Lento W, Ito T, Zhao C, et al. (2014) Loss of β-catenin triggers oxidative stress and impairs hematopoietic regeneration. Genes & Development. 28: 995-1004 |
| Lee CL, Lento WE, Castle KD, et al. (2014) Inhibiting glycogen synthase kinase-3 mitigates the hematopoietic acute radiation syndrome in mice. Radiation Research. 181: 445-51 |
| Lento W, Huang W, Doan P, et al. (2014) Calcium Calmodulin Dependent Kinase Kinase 2 Regulates Hematopoietic Stem Cell Regeneration and Quiescence Blood. 124: 1571-1571 |
| Tarrant TK, Billard MJ, Timoshchenko RG, et al. (2013) G protein-coupled receptor kinase-3-deficient mice exhibit WHIM syndrome features and attenuated inflammatory responses. Journal of Leukocyte Biology. 94: 1243-51 |
| Lento W, Congdon K, Voermans C, et al. (2013) Wnt signaling in normal and malignant hematopoiesis. Cold Spring Harbor Perspectives in Biology. 5 |
| Pei Y, Brun SN, Markant SL, et al. (2012) WNT signaling increases proliferation and impairs differentiation of stem cells in the developing cerebellum. Development (Cambridge, England). 139: 1724-33 |