John A. Dlugosz, Ph.D.

Affiliations: 
2001 University of Toronto, Toronto, ON, Canada 
Area:
Cell Biology
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"John Dlugosz"

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Catharine I. Whiteside grad student 2001 University of Toronto
 (Interaction between mesangial cell signaling and cytoskeletal function and high glucose in response to endothelin -1 and mechanical stretch.)
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Publications

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Whiteside CI, Dlugosz JA. (2002) Mesangial cell protein kinase C isozyme activation in the diabetic milieu. American Journal of Physiology. Renal Physiology. 282: F975-80
Dlugosz JA, Munk S, Ispanovic E, et al. (2002) Mesangial cell filamentous actin disassembly and hypocontractility in high glucose are mediated by PKC-zeta. American Journal of Physiology. Renal Physiology. 282: F151-63
Dlugosz JA, Munk S, Kapor-Drezgic J, et al. (2000) Stretch-induced mesangial cell ERK1/ERK2 activation is enhanced in high glucose by decreased dephosphorylation. American Journal of Physiology. Renal Physiology. 279: F688-97
Dlugosz JA, Munk S, Zhou X, et al. (1998) Endothelin-1-induced mesangial cell contraction involves activation of protein kinase C-α, -δ, and -ε. American Journal of Physiology. Renal Physiology. 275: F423-F432
Dlugosz JA, Munk S, Zhou X, et al. (1998) Endothelin-1-induced mesangial cell contraction involves activation of protein kinase C-alpha, -delta, and -epsilon. The American Journal of Physiology. 275: F423-32
Babazono T, Kapor-Drezgic J, Dlugosz JA, et al. (1998) Altered expression and subcellular localization of diacylglycerol-sensitive protein kinase C isoforms in diabetic rat glomerular cells. Diabetes. 47: 668-76
Zhou X, Li C, Dlugosz J, et al. (1997) Mesangial cell actin disassembly in high glucose mediated by protein kinase C and the polyol pathway. Kidney International. 51: 1797-808
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