John A. Dlugosz, Ph.D.
Affiliations: | 2001 | University of Toronto, Toronto, ON, Canada |
Area:
Cell BiologyGoogle:
"John Dlugosz"Parents
Sign in to add mentorCatharine I. Whiteside | grad student | 2001 | University of Toronto | |
(Interaction between mesangial cell signaling and cytoskeletal function and high glucose in response to endothelin -1 and mechanical stretch.) |
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Publications
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Whiteside CI, Dlugosz JA. (2002) Mesangial cell protein kinase C isozyme activation in the diabetic milieu. American Journal of Physiology. Renal Physiology. 282: F975-80 |
Dlugosz JA, Munk S, Ispanovic E, et al. (2002) Mesangial cell filamentous actin disassembly and hypocontractility in high glucose are mediated by PKC-zeta. American Journal of Physiology. Renal Physiology. 282: F151-63 |
Dlugosz JA, Munk S, Kapor-Drezgic J, et al. (2000) Stretch-induced mesangial cell ERK1/ERK2 activation is enhanced in high glucose by decreased dephosphorylation. American Journal of Physiology. Renal Physiology. 279: F688-97 |
Dlugosz JA, Munk S, Zhou X, et al. (1998) Endothelin-1-induced mesangial cell contraction involves activation of protein kinase C-α, -δ, and -ε. American Journal of Physiology. Renal Physiology. 275: F423-F432 |
Dlugosz JA, Munk S, Zhou X, et al. (1998) Endothelin-1-induced mesangial cell contraction involves activation of protein kinase C-alpha, -delta, and -epsilon. The American Journal of Physiology. 275: F423-32 |
Babazono T, Kapor-Drezgic J, Dlugosz JA, et al. (1998) Altered expression and subcellular localization of diacylglycerol-sensitive protein kinase C isoforms in diabetic rat glomerular cells. Diabetes. 47: 668-76 |
Zhou X, Li C, Dlugosz J, et al. (1997) Mesangial cell actin disassembly in high glucose mediated by protein kinase C and the polyol pathway. Kidney International. 51: 1797-808 |