Daniel Durocher
Affiliations: | Molecular and Medical Genetics | University of Toronto, Toronto, ON, Canada |
Area:
Genetics, Cell Biology, Molecular Biology, Oncology, ImmunologyGoogle:
"Daniel Durocher"Parents
Sign in to add mentorStephen P. Jackson | post-doc | (Neurotree) | |
Michael B. Yaffe | post-doc | MIT (Chemistry Tree) |
Children
Sign in to add traineeMichael Downey | grad student | University of Toronto (Chemistry Tree) | |
ManTek Yeung | grad student | 2012 | University of Toronto |
Stephanie Panier | grad student | 2013 | University of Toronto |
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Publications
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Fradet-Turcotte A, Canny MD, Escribano-Díaz C, et al. (2025) Editorial Expression of Concern: 53BP1 is a reader of the DNA-damage-induced H2A Lys 15 ubiquitin mark. Nature |
Nakada S, Chen GI, Gingras AC, et al. (2025) Author Correction: PP4 is a γH2AX phosphatase required for recovery from the DNA damage checkpoint. Embo Reports |
Feng S, Liu K, Shang J, et al. (2024) Profound synthetic lethality between SMARCAL1 and FANCM. Molecular Cell |
Cho T, Hoeg L, Setiaputra D, et al. (2024) NFATC2IP is a mediator of SUMO-dependent genome integrity. Genes & Development |
Zhao Y, Tabet D, Rubio Contreras D, et al. (2023) Genome-scale mapping of DNA damage suppressors through phenotypic CRISPR-Cas9 screens. Molecular Cell |
Sifri C, Hoeg L, Durocher D, et al. (2023) An AlphaFold2 map of the 53BP1 pathway identifies a direct SHLD3-RIF1 interaction critical for shieldin activity. Embo Reports. e56834 |
De Marco Zompit M, Esteban MT, Mooser C, et al. (2022) The CIP2A-TOPBP1 complex safeguards chromosomal stability during mitosis. Nature Communications. 13: 4143 |
Setiaputra D, Escribano-Díaz C, Reinert JK, et al. (2022) RIF1 acts in DNA repair through phosphopeptide recognition of 53BP1. Molecular Cell |
Adam S, Rossi SE, Moatti N, et al. (2021) The CIP2A-TOPBP1 axis safeguards chromosome stability and is a synthetic lethal target for BRCA-mutated cancer. Nature Cancer. 2: 1357-1371 |
Feng Y, Li C, Stewart JA, et al. (2021) FAM72A antagonizes UNG2 to promote mutagenic repair during antibody maturation. Nature |