Richard B. Robinson

Affiliations: 
Pharmacology and Molecular Signaling Columbia University, New York, NY 
Google:
"Richard Robinson"
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Robinson RB, Dun W, Boyden PA. (2020) Autonomic modulation of sinoatrial node: Role of pacemaker current and calcium sensitive adenylyl cyclase isoforms. Progress in Biophysics and Molecular Biology
Brown K, Legros S, Ortega FA, et al. (2017) Overexpression of Map3k7 activates sinoatrial node-like differentiation in mouse ES-derived cardiomyocytes. Plos One. 12: e0189818
Eng G, Lee BW, Protas L, et al. (2016) Autonomous beating rate adaptation in human stem cell-derived cardiomyocytes. Nature Communications. 7: 10312
Boyden PA, Dun W, Robinson RB. (2016) Cardiac Purkinje fibers and arrhythmias; The GK Moe Award Lecture 2015. Heart Rhythm. 13: 1172-81
Harleton E, Besana A, Chandra P, et al. (2015) TASK-1 current is inhibited by phosphorylation during human and canine chronic atrial fibrillation. American Journal of Physiology. Heart and Circulatory Physiology. 308: H126-34
Boink GJ, Robinson RB. (2014) Gene therapy for restoring heart rhythm. Journal of Cardiovascular Pharmacology and Therapeutics. 19: 426-38
Morris GM, D'Souza A, Dobrzynski H, et al. (2013) Characterization of a right atrial subsidiary pacemaker and acceleration of the pacing rate by HCN over-expression. Cardiovascular Research. 100: 160-9
Nawathe PA, Kryukova Y, Oren RV, et al. (2013) An LQTS6 MiRP1 mutation suppresses pacemaker current and is associated with sinus bradycardia. Journal of Cardiovascular Electrophysiology. 24: 1021-7
Boink GJ, Duan L, Nearing BD, et al. (2013) HCN2/SkM1 gene transfer into canine left bundle branch induces stable, autonomically responsive biological pacing at physiological heart rates. Journal of the American College of Cardiology. 61: 1192-201
Harleton E, Besana A, Comas GM, et al. (2013) Ability to induce atrial fibrillation in the peri-operative period is associated with phosphorylation-dependent inhibition of TWIK protein-related acid-sensitive potassium channel 1 (TASK-1). The Journal of Biological Chemistry. 288: 2829-38
See more...