Terrence N Wong, MD PhD
Affiliations: | 2012-2019 | Medicine | Washington University, Saint Louis, St. Louis, MO |
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"Terrence Wong"Parents
Sign in to add mentor| David M. Lemal | research assistant | 1999-2001 | Dartmouth (Chemistry Tree) |
| Tao Pan | grad student | 2003-2007 | Chicago (Chemistry Tree) |
| Daniel C. Link | post-doc | 2012-2019 | Washington University |
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Publications
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| Chen S, Wang Q, Yu H, et al. (2020) Author Correction: Mutant p53 drives clonal hematopoiesis through modulating epigenetic pathway. Nature Communications. 11: 3856 |
| Chen S, Wang Q, Yu H, et al. (2019) Mutant p53 drives clonal hematopoiesis through modulating epigenetic pathway. Nature Communications. 10: 5649 |
| Berrien-Elliott MM, Sun Y, Neal C, et al. (2019) MicroRNA-142 Is Critical for the Homeostasis and Function of Type 1 Innate Lymphoid Cells. Immunity |
| Trissal MC, Wong TN, Yao JC, et al. (2018) MIR142 loss-of-function mutations derepress ASH1L to increase HOXA gene expression and promote leukemogenesis. Cancer Research |
| Wong TN, Miller CA, Jotte MRM, et al. (2018) Cellular stressors contribute to the expansion of hematopoietic clones of varying leukemic potential. Nature Communications. 9: 455 |
| Wong TN, Link DC. (2017) miR-125b promotes leukemogenesis via VEGFA. Blood. 129: 1409-1410 |
| Wong TN, Miller CA, Jotte MR, et al. (2017) Cellular Stressors Contribute to the Expansion of Multiple Hematopoietic Clones of Varying Leukemic Potential Blood. 130: 469-469 |
| Yao J, Wong TN, Trissal M, et al. (2017) Abstract 3042:MIR142loss-of-function mutations promote leukemogenesis through derepression ofASH1Lresulting in increasedHOXgene expression Cancer Research. 77: 3042-3042 |
| Yao J, Wong TN, Trissal M, et al. (2016) MIR142 Loss-of-Function Mutations Promote Leukemogenesis through Derepression of ASH1L Resulting in Increased HOX Gene Expression Blood. 128: 2718-2718 |
| Wong TN, Miller CA, Klco JM, et al. (2015) Rapid expansion of pre-existing non-leukemic hematopoietic clones frequently follows induction therapy for de novo AML. Blood |