Shraddha R. Desai, Ph.D. - Publications

Affiliations: 
2011 Chemistry University of South Florida, Tampa, FL, United States 
Area:
Biochemistry, Cell Biology

14 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2015 Bowie M, Pilie P, Wulfkuhle J, Lem S, Hoffman A, Desai S, Petricoin E, Carter A, Ambrose A, Seewaldt V, Yu D, Ibarra Drendall C. Fluoxetine induces cytotoxic endoplasmic reticulum stress and autophagy in triple negative breast cancer. World Journal of Clinical Oncology. 6: 299-311. PMID 26677444 DOI: 10.5306/Wjco.V6.I6.299  0.318
2015 Desai SR, Dietze EC, Skaar D, Jirtle RL, Seewaldt VL. Abstract PR11: Loss of imprinting: Tying prenatal diet to the aggressive biology of triple-negative breast cancer Cancer Epidemiology, Biomarkers & Prevention. 24. DOI: 10.1158/1538-7755.Disp14-Pr11  0.354
2014 McCray AN, Desai S, Acevedo-Duncan M. The interruption of PKC-ι signaling and TRAIL combination therapy against glioblastoma cells. Neurochemical Research. 39: 1691-701. PMID 24965532 DOI: 10.1007/S11064-014-1361-8  0.719
2014 Desai S, Skaar D, Dietze E, Ambrose A, Jirtle R, Seewaldt V. Abstract C65: Loss of imprinting of KCNK9 in African American women with triple-negative breast cancer Cancer Epidemiology, Biomarkers & Prevention. 23. DOI: 10.1158/1538-7755.Disp13-C65  0.34
2014 Seewaldt V, Skaar D, Dietze E, Desai S, Jirtle R. Abstract 28: Chemoprevention strategies to target abnormal imprinting in high-risk African American women who do or do not carry a BRCA1 mutation Cancer Research. 74: 28-28. DOI: 10.1158/1538-7445.Cansusc14-28  0.332
2013 McCray AN, Desai S, Acevedo-Duncan M. Abstract 2952: The PKC-iota inhibitor ICA-1 combined with TRAIL for glioblastoma therapy. Cancer Research. 73: 2952-2952. DOI: 10.1158/1538-7445.Am2013-2952  0.69
2012 Desai SR, Pillai PP, Patel RS, McCray AN, Win-Piazza HY, Acevedo-Duncan ME. Regulation of Cdk7 activity through a phosphatidylinositol (3)-kinase/PKC-ι-mediated signaling cascade in glioblastoma. Carcinogenesis. 33: 10-9. PMID 22021906 DOI: 10.1093/Carcin/Bgr231  0.789
2012 Desai S, Patel R, McCray AN, Pillai P, Acevedo-Duncan M. Abstract 3760: A novel PKC-iota inhibitor, ICA-1 abrogates cell proliferation in breast cancer Cancer Research. 72: 3760-3760. DOI: 10.1158/1538-7445.Am2012-3760  0.744
2011 Desai S, Pillai P, Win-Piazza H, Acevedo-Duncan M. PKC-3 promotes glioblastoma cell survival by phosphorylating and inhibiting BAD through a phosphatidylinositol 3-kinase pathway Biochimica Et Biophysica Acta - Molecular Cell Research. 1813: 1190-1197. PMID 21419810 DOI: 10.1016/J.Bbamcr.2011.03.007  0.788
2011 Pillai P, Desai S, Patel R, Sajan M, Farese R, Ostrov D, Acevedo-Duncan M. A novel PKC-ι inhibitor abrogates cell proliferation and induces apoptosis in neuroblastoma. The International Journal of Biochemistry & Cell Biology. 43: 784-94. PMID 21315177 DOI: 10.1016/J.Biocel.2011.02.002  0.797
2011 Desai S, Acevedo-Duncan M. Abstract 7: PKC-ι promotes glioblastoma cell survival by phosphorylating and inhibiting BAD through a phosphotidylinositol 3-kinase pathway Cancer Research. 71: 7-7. DOI: 10.1158/1538-7445.Am2011-7  0.794
2011 Desai S, Acevedo-Duncan M. Abstract 2974: Regulation of cyclin dependent kinase 7 through a phosphotidylinositol 3-kinase/PKC-ι mediated cell proliferation cascade Cancer Research. 71: 2974-2974. DOI: 10.1158/1538-7445.Am2011-2974  0.788
2011 Pillai P, Desai S, Patel R, Sajan M, Farese R, Ostrov D, Acevedo-Duncan M. Erratum to “A novel PKC-ι inhibitor abrogates cell proliferation and induces apoptosis in neuroblastoma” [Int. J. Biochem. Cell Biol. 43 (5) (2011) 784–794] The International Journal of Biochemistry & Cell Biology. 43: 1412. DOI: 10.1016/J.Biocel.2011.05.015  0.76
2010 Desai S, Acevedo-Duncan M. Abstract LB-113: PKC-iota induces uncontrolled glioblastoma cell survival by modulating proapoptotic function of Bad Cancer Research. 70. DOI: 10.1158/1538-7445.Am10-Lb-113  0.785
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