| Year |
Citation |
Score |
| 2014 |
Payne GA, Tune JD, Knudson JD. Leptin-induced endothelial dysfunction: A target for therapeutic interventions Current Pharmaceutical Design. 20: 603-608. PMID 23688011 DOI: 10.2174/13816128113199990017 |
0.799 |
|
| 2013 |
Lowry AW, Olabiyi OO, Adachi I, Moodie DS, Knudson JD. Coronary artery anatomy in congenital heart disease. Congenital Heart Disease. 8: 187-202. PMID 23601059 DOI: 10.1111/chd.12067 |
0.336 |
|
| 2008 |
Knudson JD, Payne GA, Borbouse L, Tune JD. Leptin and mechanisms of endothelial dysfunction and cardiovascular disease Current Hypertension Reports. 10: 434-439. PMID 18959828 DOI: 10.1007/S11906-008-0082-2 |
0.728 |
|
| 2008 |
Dick GM, Bratz IN, Borbouse L, Payne GA, Dincer UD, Knudson JD, Rogers PA, Tune JD. Voltage-dependent K+ channels regulate the duration of reactive hyperemia in the canine coronary circulation. American Journal of Physiology. Heart and Circulatory Physiology. 294: H2371-81. PMID 18375717 DOI: 10.1152/Ajpheart.01279.2007 |
0.664 |
|
| 2008 |
Zhang C, Rogers PA, Merkus D, Muller-Delp JM, Tiefenbacher CP, Potter B, Knudson JD, Rocic P, Chilian WM. Regulation of Coronary Microvascular Resistance in Health and Disease Microcirculation. 521-549. DOI: 10.1016/B978-0-12-374530-9.00014-0 |
0.425 |
|
| 2007 |
Knudson JD, Dincer UD, Bratz IN, Sturek M, Dick GM, Tune JD. Mechanisms of coronary dysfunction in obesity and insulin resistance. Microcirculation (New York, N.Y. : 1994). 14: 317-38. PMID 17613805 DOI: 10.1080/10739680701282887 |
0.803 |
|
| 2007 |
Knudson JD, Dick GM, Tune JD. Adipokines and coronary vasomotor dysfunction. Experimental Biology and Medicine (Maywood, N.J.). 232: 727-36. PMID 17526764 DOI: 10.3181/00379727-232-2320727 |
0.689 |
|
| 2006 |
Saitoh SI, Zhang C, Tune JD, Potter B, Kiyooka T, Rogers PA, Knudson JD, Dick GM, Swafford A, Chilian WM. Hydrogen peroxide: A feed-forward dilator that couples myocardial metabolism to coronary blood flow Arteriosclerosis, Thrombosis, and Vascular Biology. 26: 2614-2621. PMID 17023676 DOI: 10.1161/01.Atv.0000249408.55796.Da |
0.643 |
|
| 2006 |
Dincer UD, Araiza AG, Knudson JD, Molina PE, Tune JD. Sensitization of coronary alpha-adrenoceptor vasoconstriction in the prediabetic metabolic syndrome. Microcirculation (New York, N.Y. : 1994). 13: 587-95. PMID 16990217 DOI: 10.1080/10739680600885228 |
0.748 |
|
| 2006 |
Dick GM, Katz PS, Farias M, Morris M, James J, Knudson JD, Tune JD. Resistin impairs endothelium-dependent dilation to bradykinin, but not acetylcholine, in the coronary circulation. American Journal of Physiology. Heart and Circulatory Physiology. 291: H2997-3002. PMID 16905596 DOI: 10.1152/Ajpheart.01035.2005 |
0.715 |
|
| 2006 |
Dincer UD, Araiza A, Knudson JD, Shao CH, Bidasee KR, Tune JD. Dysfunction of cardiac ryanodine receptors in the metabolic syndrome. Journal of Molecular and Cellular Cardiology. 41: 108-14. PMID 16793060 DOI: 10.1016/J.Yjmcc.2006.04.018 |
0.716 |
|
| 2006 |
Rogers PA, Dick GM, Knudson JD, Focardi M, Bratz IN, Swafford AN, Saitoh S, Tune JD, Chilian WM. H2O2-induced redox-sensitive coronary vasodilation is mediated by 4-aminopyridine-sensitive K+ channels. American Journal of Physiology. Heart and Circulatory Physiology. 291: H2473-82. PMID 16751285 DOI: 10.1152/Ajpheart.00172.2006 |
0.791 |
|
| 2006 |
Knudson JD, Rogers PA, Dincer UD, Bratz IN, Araiza AG, Dick GM, Tune JD. Coronary vasomotor reactivity to endothelin-1 in the prediabetic metabolic syndrome. Microcirculation (New York, N.Y. : 1994). 13: 209-18. PMID 16627363 DOI: 10.1080/10739680600556894 |
0.772 |
|
| 2005 |
Knudson JD, Dincer UD, Dick GM, Shibata H, Akahane R, Saito M, Tune JD. Leptin resistance extends to the coronary vasculature in prediabetic dogs and provides a protective adaptation against endothelial dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. 289: H1038-46. PMID 15894577 DOI: 10.1152/Ajpheart.00244.2005 |
0.805 |
|
| 2005 |
Knudson JD, Dincer UD, Zhang C, Swafford AN, Koshida R, Picchi A, Focardi M, Dick GM, Tune JD. Leptin receptors are expressed in coronary arteries, and hyperleptinemia causes significant coronary endothelial dysfunction. American Journal of Physiology. Heart and Circulatory Physiology. 289: H48-56. PMID 15749744 DOI: 10.1152/Ajpheart.01159.2004 |
0.805 |
|
| 2005 |
Zhang C, Knudson JD, Setty S, Araiza A, Dincer UD, Kuo L, Tune JD. Coronary arteriolar vasoconstriction to angiotensin II is augmented in prediabetic metabolic syndrome via activation of AT1 receptors. American Journal of Physiology. Heart and Circulatory Physiology. 288: H2154-62. PMID 15653764 DOI: 10.1152/Ajpheart.00987.2004 |
0.776 |
|
| 2005 |
Swafford AN, Bratz IN, Knudson JD, Rogers PA, Timmerman JM, Tune JD, Dick GM. C-reactive protein does not relax vascular smooth muscle: effects mediated by sodium azide in commercially available preparations. American Journal of Physiology. Heart and Circulatory Physiology. 288: H1786-95. PMID 15563529 DOI: 10.1152/Ajpheart.00996.2004 |
0.783 |
|
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