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Paul F. Pilch

Affiliations: 
Boston University, Boston, MA, United States 
Area:
Molecular Biology, Biochemistry
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Parents

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Michael P. Czech post-doc

Children

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Jongsoon Lee grad student (Physiology Academic Tree)
Lori L. Tortorella grad student 2001 Boston University
Danielle N. Gross grad student 2004 Boston University
Zidong Zhang grad student 2008 Boston University
Michael R. Breen grad student 2011 Boston University
Brian Honeyman grad student 2015 Boston University

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Publications

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Wang H, Pilch PF, Liu L. (2019) Cavin-1/PTRF mediates insulin-dependent focal adhesion remodeling and ameliorates high-fat diet-induced inflammatory responses in mice. The Journal of Biological Chemistry
Williams JJL, Alotaiq N, Mullen W, et al. (2018) Interaction of suppressor of cytokine signalling 3 with cavin-1 links SOCS3 function and cavin-1 stability. Nature Communications. 9: 168
Ding SY, Liu L, Pilch PF. (2017) Muscular dystrophy in PTFR/cavin-1 null mice. Jci Insight. 2: e91023
Jedrychowski MP, Liu L, Laflamme CJ, et al. (2015) Adiporedoxin, an upstream regulator of ER oxidative folding and protein secretion in adipocytes. Molecular Metabolism. 4: 758-770
Lo HP, Nixon SJ, Hall TE, et al. (2015) The caveolin-cavin system plays a conserved and critical role in mechanoprotection of skeletal muscle. The Journal of Cell Biology. 210: 833-49
Scheller EL, Doucette CR, Learman BS, et al. (2015) Region-specific variation in the properties of skeletal adipocytes reveals regulated and constitutive marrow adipose tissues. Nature Communications. 6: 7808
Liu L, Hansen CG, Honeyman BJ, et al. (2014) Cavin-3 knockout mice show that cavin-3 is not essential for caveolae formation, for maintenance of body composition, or for glucose tolerance. Plos One. 9: e102935
Ding SY, Lee MJ, Summer R, et al. (2014) Pleiotropic effects of cavin-1 deficiency on lipid metabolism. The Journal of Biological Chemistry. 289: 8473-83
Fernández-Rojo MA, Gongora M, Fitzsimmons RL, et al. (2013) Caveolin-1 is necessary for hepatic oxidative lipid metabolism: evidence for crosstalk between caveolin-1 and bile acid signaling. Cell Reports. 4: 238-47
Scotti E, Calamai M, Goulbourne CN, et al. (2013) IDOL stimulates clathrin-independent endocytosis and multivesicular body-mediated lysosomal degradation of the low-density lipoprotein receptor. Molecular and Cellular Biology. 33: 1503-14
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