Christopher D. Wiley, Ph.D.
Affiliations: | 2002-2008 | University of California, Irvine, Irvine, CA | |
| 2008-2020 | Buck Institute for Research on Aging | ||
| 2020- | Jean Mayer USDA Human Nutrition Research Center on Aging at Tufts University, Boston, MA, United States |
Area:
Genetics, Anatomy Biology, Molecular BiologyGoogle:
"Christopher Wiley"Parents
Sign in to add mentor| Paul B. Vrana | grad student | 2008 | UC Irvine | |
| (Genetic and epigenetic analysis of parent-of-origin effects in mammals.) | ||||
| Judith Campisi | post-doc | 2008-2018 | Buck Institute for Research on Aging (Cell Biology Tree) | |
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Publications
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| Mogck BA, Jezak ST, Wiley CD. (2024) Mitochondria-Targeted Catalase Does Not Suppress Development of Cellular Senescence during Aging. Biomedicines. 12 |
| Kuehnemann C, Wiley CD. (2023) Senescent cells at the crossroads of aging, disease, and tissue homeostasis. Aging Cell. e13988 |
| Faggioli F, Velarde MC, Wiley CD. (2023) Cellular Senescence, a Novel Area of Investigation for Metastatic Diseases. Cells. 12 |
| Kuehnemann C, Hughes JB, Desprez PY, et al. (2022) Antiretroviral protease inhibitors induce features of cellular senescence that are reversible upon drug removal. Aging Cell. e13750 |
| Wiley CD, Campisi J. (2021) The metabolic roots of senescence: mechanisms and opportunities for intervention. Nature Metabolism. 3: 1290-1301 |
| Wiley CD, Sharma R, Davis SS, et al. (2021) Oxylipin biosynthesis reinforces cellular senescence and allows detection of senolysis. Cell Metabolism |
| Covarrubias AJ, Kale A, Perrone R, et al. (2020) Author Correction: Senescent cells promote tissue NAD decline during ageing via the activation of CD38 macrophages. Nature Metabolism |
| Covarrubias AJ, Kale A, Perrone R, et al. (2020) Senescent cells promote tissue NAD decline during ageing via the activation of CD38 macrophages. Nature Metabolism. 2: 1265-1283 |
| Alimirah F, Pulido T, Valdovinos A, et al. (2020) Cellular senescence promotes skin carcinogenesis through p38MAPK and p44/p42MAPK signaling. Cancer Research |
| Kim DE, Dollé MET, Vermeij WP, et al. (2019) Deficiency in the DNA repair protein ERCC1 triggers a link between senescence and apoptosis in human fibroblasts and mouse skin. Aging Cell. e13072 |