Stephen C. Parnell, Ph.D.
Affiliations: | 2001 | University of Kansas, Lawrence, KS, United States |
Area:
Molecular Biology, Biochemistry, PathologyGoogle:
"Stephen Parnell"Mean distance: (not calculated yet)
Parents
Sign in to add mentor| James P. Calvet | grad student | 2001 | University of Kansas | |
| (Polycystin-1 is a phosphoprotein that regulates heterotrimeric G protein signaling.) | ||||
| Henrik G. Dohlman | post-doc | 2001-2006 | UNC Chapel Hill (Physics Tree) | |
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Publications
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| Maser RL, Calvet JP, Parnell SC. (2022) The GPCR properties of polycystin-1- A new paradigm. Frontiers in Molecular Biosciences. 9: 1035507 |
| Lea WA, McGreal K, Sharma M, et al. (2020) Analysis of the polycystin complex (PCC) in human urinary exosome-like vesicles (ELVs). Scientific Reports. 10: 1500 |
| Raman A, Parnell SC, Zhang Y, et al. (2018) Periostin overexpression in collecting ducts accelerates renal cyst growth and fibrosis in polycystic kidney disease. American Journal of Physiology. Renal Physiology |
| Lea WA, Parnell SC, Wallace DP, et al. (2018) Human-Specific Abnormal Alternative Splicing of Wild-Type Induces Premature Termination of Polycystin-1. Journal of the American Society of Nephrology : Jasn |
| Parnell SC, Magenheimer BS, Maser RL, et al. (2018) A Mutation Affecting Polycystin-1 Mediated Heterotrimeric G-Protein Signaling Causes PKD. Human Molecular Genetics |
| Raman A, Reif GA, Dai Y, et al. (2017) Integrin-Linked Kinase Signaling Promotes Cyst Growth and Fibrosis in Polycystic Kidney Disease. Journal of the American Society of Nephrology : Jasn |
| Hao N, Yildirim N, Nagiec MJ, et al. (2012) Combined computational and experimental analysis reveals mitogen-activated protein kinase-mediated feedback phosphorylation as a mechanism for signaling specificity. Molecular Biology of the Cell. 23: 3899-910 |
| Parnell SC, Puri S, Wallace DP, et al. (2012) Protein phosphatase-1α interacts with and dephosphorylates polycystin-1. Plos One. 7: e36798 |
| Zeller CE, Parnell SC, Dohlman HG. (2007) The RACK1 ortholog Asc1 functions as a G-protein beta subunit coupled to glucose responsiveness in yeast. The Journal of Biological Chemistry. 282: 25168-76 |
| Hao N, Behar M, Parnell SC, et al. (2007) A systems-biology analysis of feedback inhibition in the Sho1 osmotic-stress-response pathway. Current Biology : Cb. 17: 659-67 |