Daniel C. Bartos, Ph.D.

Affiliations: 
2013 Physiology University of Kentucky, Lexington, KY 
Area:
Physiology Biology, General Biophysics
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Brian P. Delisle grad student 2013 University of Kentucky
 (Mechanistic basis for atrial and ventricular arrhythmias caused by KCNQ1 mutations.)
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Publications

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Kekenes-Huskey PM, Burgess DE, Sun B, et al. (2022) Mutation-Specific Differences in Kv7.1 () and Kv11.1 () Channel Dysfunction and Long QT Syndrome Phenotypes. International Journal of Molecular Sciences. 23
Liao Z, Ginsburg KS, Bartos DC, et al. (2017) High Diastolic Sodium Influx in Heart Failure has Drug-Sensitivities Like Late Sodium Current, but Produces Inward Current at Diastolic Potentials Biophysical Journal. 112: 333a
Grandi E, Sanguinetti MC, Bartos DC, et al. (2016) Potassium channels in the heart: structure, function and regulation. The Journal of Physiology
Ackerman JP, Bartos DC, Kapplinger JD, et al. (2016) The Promise and Peril of Precision Medicine: Phenotyping Still Matters Most. Mayo Clinic Proceedings
Bartos DC, Grandi E, Ripplinger CM. (2015) Ion Channels in the Heart. Comprehensive Physiology. 5: 1423-64
Schroder EA, Burgess DE, Zhang X, et al. (2015) The cardiomyocyte molecular clock regulates the circadian expression of Kcnh2 and contributes to ventricular repolarization. Heart Rhythm : the Official Journal of the Heart Rhythm Society. 12: 1306-14
Bartos DC, Giudicessi JR, Tester DJ, et al. (2014) A KCNQ1 mutation contributes to the concealed type 1 long QT phenotype by limiting the Kv7.1 channel conformational changes associated with protein kinase A phosphorylation. Heart Rhythm : the Official Journal of the Heart Rhythm Society. 11: 459-68
Smith JL, Reloj AR, Nataraj PS, et al. (2013) Pharmacological correction of long QT-linked mutations in KCNH2 (hERG) increases the trafficking of Kv11.1 channels stored in the transitional endoplasmic reticulum. American Journal of Physiology. Cell Physiology. 305: C919-30
Crotti L, Tester DJ, White WM, et al. (2013) Long QT syndrome-associated mutations in intrauterine fetal death. Jama. 309: 1473-82
McBride CM, Smith AM, Smith JL, et al. (2013) Mechanistic basis for type 2 long QT syndrome caused by KCNH2 mutations that disrupt conserved arginine residues in the voltage sensor. The Journal of Membrane Biology. 246: 355-64
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