Vernon M. Ingram

Biology Massachusetts Institute of Technology, Cambridge, MA, United States 
"Vernon M. Ingram"

(1924 - 2006)

Mean distance: 9.25


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John R. Bankston research assistant 2001-2003 MIT (Neurotree)
Antony Stretton grad student (Neurotree)
Antony O. W. Stretton grad student (Neurotree)
Corrado Baglioni post-doc 1961 MIT
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Ahmad A, Muzaffar M, Ingram VM. (2009) Ca2+, within the physiological concentrations, selectively accelerates Aβ42 fibril formation and not Aβ40 in vitro Biochimica Et Biophysica Acta - Proteins and Proteomics. 1794: 1537-1548
Colby DW, Cassady JP, Lin GC, et al. (2006) Stochastic kinetics of intracellular huntingtin aggregate formation. Nature Chemical Biology. 2: 319-23
Dahlgren PR, Karymov MA, Bankston J, et al. (2005) Atomic force microscopy analysis of the Huntington protein nanofibril formation. Disease-a-Month : Dm.. 51: 374-385
Ingram VM. (2005) The role of Alzheimer Abeta peptides in ion transport across cell membranes. Sub-Cellular Biochemistry. 38: 339-349
Colby DW, Chu Y, Cassady JP, et al. (2004) Potent inhibition of huntingtin aggregation and cytotoxicity by a disulfide bond-free single-domain intracellular antibody. Proceedings of the National Academy of Sciences of the United States of America. 101: 17616-21
Blanchard BJ, Chen A, Rozeboom LM, et al. (2004) Efficient reversal of Alzheimer's disease fibril formation and elimination of neurotoxicity by a small molecule. Proceedings of the National Academy of Sciences of the United States of America. 101: 14326-32
Colby DW, Garg P, Holden T, et al. (2004) Development of a human light chain variable domain (V(L)) intracellular antibody specific for the amino terminus of huntingtin via yeast surface display. Journal of Molecular Biology. 342: 901-12
Ingram VM. (2004) Sickle-cell anemia hemoglobin: The molecular biology of the first "molecular disease" - The crucial importance of serendipity Genetics. 167: 1-7
Ingram V. (2003) Alzheimer's disease American Scientist. 91: 312-321
Blanchard BJ, Stockwell BR, Ingram VM. (2002) Eliminating membrane depolarization caused by the Alzheimer peptide Abeta(1-42, aggr.). Biochemical and Biophysical Research Communications. 293: 1204-8
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