Year |
Citation |
Score |
2022 |
Omkar S, Wani TH, Zheng B, Mitchem MM, Truman AW. The APE2 Exonuclease Is a Client of the Hsp70-Hsp90 Axis in Yeast and Mammalian Cells. Biomolecules. 12. PMID 35883419 DOI: 10.3390/biom12070864 |
0.359 |
|
2021 |
Chakrabarty A, Surendran S, Bhola NE, Mishra VS, Wani TH, Baghel KS, Arteaga CL, Garg R, Chowdhury G. Corrigendum to: The H1047R PIK3CA oncogene induces a senescence-like state, pleiotropy and acute HSP90 dependency in HER2+ mammary epithelial cells. Carcinogenesis. PMID 34363390 DOI: 10.1093/carcin/bgab065 |
0.653 |
|
2021 |
Wani TH, Chowdhury G, Chakrabarty A. Generation of reactive oxygen species is the primary mode of action and cause of survivin suppression by sepantronium bromide (YM155). Rsc Medicinal Chemistry. 12: 566-578. PMID 34046628 DOI: 10.1039/d0md00383b |
0.689 |
|
2019 |
Chakrabarty A, Surendran S, Bhola NE, Mishra VS, Wani TH, Baghel KS, Arteaga CL, Garg R, Chowdhury G. The H1047R PIK3CA oncogene induces a senescence-like state, pleiotropy and acute HSP90 dependency in HER2+ mammary epithelial cells. Carcinogenesis. PMID 31219154 DOI: 10.1093/Carcin/Bgz118 |
0.67 |
|
2018 |
Wani TH, Surendran S, Mishra VS, Chaturvedi J, Chowdhury G, Chakrabarty A. Adaptation to chronic exposure to sepantronium bromide (YM155), a prototypical survivin suppressant is due to persistent DNA damage-response in breast cancer cells. Oncotarget. 9: 33589-33600. PMID 30323901 DOI: 10.18632/Oncotarget.26096 |
0.697 |
|
2018 |
Wani TH, Surendran S, Jana A, Chakrabarty A, Chowdhury G. The Quinone Based Antitumor Agent Sepantronium Bromide (YM155) Causes Oxygen Independent Redox Activated Oxidative DNA Damage. Chemical Research in Toxicology. PMID 29897742 DOI: 10.1021/Acs.Chemrestox.8B00094 |
0.678 |
|
2017 |
Wani TH, Chakrabarty A, Shibata N, Yamazaki H, Guengerich FP, Chowdhury G. The Dihydroxy Metabolite of the Teratogen Thalidomide Causes Oxidative DNA Damage. Chemical Research in Toxicology. PMID 28745489 DOI: 10.1021/Acs.Chemrestox.7B00127 |
0.683 |
|
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