Christopher H. S. McIntosh
Affiliations: | University of British Columbia, Vancouver, Vancouver, BC, Canada |
Area:
Animal Physiology Biology, Cell BiologyWebsite:
https://cps.med.ubc.ca/faculty1/mcintosh/Google:
"Christopher H. S. McIntosh"Bio:
https://diabetes.ubc.ca/primary-members/dr-christopher-h-s-mcintosh/
https://books.google.com/books?id=yO4aAQAAMAAJ
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1177755/
Children
Sign in to add trainee| Jan A. Ehses | grad student | 2003 | UBC |
| Simon A. Hinke | grad student | 2003 | UBC |
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Publications
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| Pederson RA, McIntosh CH. (2016) Discovery of gastric inhibitory polypeptide and its subsequent fate: Personal reflections. Journal of Diabetes Investigation. 7: 4-7 |
| Kim SJ, Nian C, McIntosh CH. (2013) Resistin knockout mice exhibit impaired adipocyte glucose-dependent insulinotropic polypeptide receptor (GIPR) expression. Diabetes. 62: 471-7 |
| McIntosh CH, Widenmaier S, Kim SJ. (2012) Glucose-dependent insulinotropic polypeptide signaling in pancreatic β-cells and adipocytes. Journal of Diabetes Investigation. 3: 96-106 |
| Kim SJ, Nian C, Karunakaran S, et al. (2012) GIP-overexpressing mice demonstrate reduced diet-induced obesity and steatosis, and improved glucose homeostasis. Plos One. 7: e40156 |
| Whitaker GM, Lynn FC, McIntosh CH, et al. (2012) Regulation of GIP and GLP1 receptor cell surface expression by N-glycosylation and receptor heteromerization. Plos One. 7: e32675 |
| Kim SJ, Nian C, McIntosh CH. (2011) Adipocyte expression of the glucose-dependent insulinotropic polypeptide receptor involves gene regulation by PPARγ and histone acetylation. Journal of Lipid Research. 52: 759-70 |
| McIntosh CH, Widenmaier S, Kim SJ. (2010) Pleiotropic actions of the incretin hormones. Vitamins and Hormones. 84: 21-79 |
| Kim SJ, Nian C, McIntosh CH. (2010) GIP increases human adipocyte LPL expression through CREB and TORC2-mediated trans-activation of the LPL gene. Journal of Lipid Research. 51: 3145-57 |
| Widenmaier SB, Kim SJ, Yang GK, et al. (2010) A GIP receptor agonist exhibits beta-cell anti-apoptotic actions in rat models of diabetes resulting in improved beta-cell function and glycemic control. Plos One. 5: e9590 |
| Gelling RW, Vuguin PM, Du XQ, et al. (2009) Pancreatic beta-cell overexpression of the glucagon receptor gene results in enhanced beta-cell function and mass. American Journal of Physiology. Endocrinology and Metabolism. 297: E695-707 |