| Year |
Citation |
Score |
| 2023 |
Corano-Scheri K, Lavine JA, Tedeschi TR, Thomson BR, Fawzi AA. Single cell transcriptomic analysis of proliferative diabetic retinopathy fibrovascular membranes reveals AEBP1 as fibrogenesis modulator. Jci Insight. PMID 37917183 DOI: 10.1172/jci.insight.172062 |
0.316 |
|
| 2017 |
Lavine JA, Farnoodian M, Wang S, Darjatmoko SR, Wright LS, Gamm DM, Ip MS, Sorenson CM, Sheibani N. β2-Adrenergic Receptor Antagonism Attenuates CNV Through Inhibition of VEGF and IL-6 Expression. Investigative Ophthalmology & Visual Science. 58: 299-308. PMID 28114591 DOI: 10.1167/Iovs.16-20204 |
0.399 |
|
| 2015 |
Lavine JA, Kibbe CR, Baan M, Sirinvaravong S, Umhoefer HM, Engler KA, Meske LM, Sacotte KA, Erhardt DP, Davis DB. Cholecystokinin expression in the β-cell leads to increased β-cell area in aged mice and protects from streptozotocin-induced diabetes and apoptosis. American Journal of Physiology. Endocrinology and Metabolism. ajpendo.00159.2015. PMID 26394663 DOI: 10.1152/Ajpendo.00159.2015 |
0.679 |
|
| 2013 |
Krautkramer KA, Linnemann AK, Fontaine DA, Whillock AL, Harris TW, Schleis GJ, Truchan NA, Marty-Santos L, Lavine JA, Cleaver O, Kimple ME, Davis DB. Tcf19 is a novel islet factor necessary for proliferation and survival in the INS-1 β-cell line. American Journal of Physiology. Endocrinology and Metabolism. 305: E600-10. PMID 23860123 DOI: 10.1152/Ajpendo.00147.2013 |
0.789 |
|
| 2013 |
Lavine JA, Sang Y, Wang S, Ip MS, Sheibani N. Attenuation of choroidal neovascularization by β(2)-adrenoreceptor antagonism. Jama Ophthalmology. 131: 376-82. PMID 23303344 DOI: 10.1001/Jamaophthalmol.2013.1476 |
0.423 |
|
| 2010 |
Lavine JA, Attie AD. Gastrointestinal hormones and the regulation of β-cell mass. Annals of the New York Academy of Sciences. 1212: 41-58. PMID 21039588 DOI: 10.1111/J.1749-6632.2010.05802.X |
0.771 |
|
| 2010 |
Davis DB, Lavine JA, Suhonen JI, Krautkramer KA, Rabaglia ME, Sperger JM, Fernandez LA, Yandell BS, Keller MP, Wang IM, Schadt EE, Attie AD. FoxM1 is up-regulated by obesity and stimulates beta-cell proliferation. Molecular Endocrinology (Baltimore, Md.). 24: 1822-34. PMID 20660304 DOI: 10.1210/Me.2010-0082 |
0.792 |
|
| 2010 |
Lavine JA, Raess PW, Stapleton DS, Rabaglia ME, Suhonen JI, Schueler KL, Koltes JE, Dawson JA, Yandell BS, Samuelson LC, Beinfeld MC, Davis DB, Hellerstein MK, Keller MP, Attie AD. Cholecystokinin is up-regulated in obese mouse islets and expands beta-cell mass by increasing beta-cell survival. Endocrinology. 151: 3577-88. PMID 20534724 DOI: 10.1210/En.2010-0233 |
0.756 |
|
| 2010 |
Lavine JA, Raess PW, Davis DB, Rabaglia ME, Presley BK, Keller MP, Beinfeld MC, Kopin AS, Newgard CB, Attie AD. Contamination with E1A-positive wild-type adenovirus accounts for species-specific stimulation of islet cell proliferation by CCK: a cautionary note. Molecular Endocrinology (Baltimore, Md.). 24: 464-7. PMID 20081104 DOI: 10.1210/Me.2009-0384 |
0.725 |
|
| 2010 |
Davis DB, Lavine JA, Suhonen JI, Krautkramer KA, Rabaglia ME, Sperger JM, Fernandez LA, Yandell BS, Keller MP, Wang I, Schadt EE, Attie AD. FoxM1 Is Up-Regulated by Obesity and Stimulates β-Cell Proliferation The Journal of Clinical Endocrinology & Metabolism. 95: 4084-4084. DOI: 10.1210/Jcem.95.8.9994 |
0.781 |
|
| 2010 |
Lavine JA, Raess PW, Davis DB, Rabaglia ME, Presley BK, Keller MP, Beinfeld MC, Kopin AS, Newgard CB, Attie AD. Overexpression of pre-pro-cholecystokinin stimulates β-cell proliferation in mouse and human islets with retention of islet function (Molecular Endocrinology (2008) 22, (2716-2728)) Molecular Endocrinology. 24: 472. |
0.744 |
|
| 2008 |
Lavine JA, Raess PW, Davis DB, Rabaglia ME, Presley BK, Keller MP, Beinfeld MC, Kopin AS, Newgard CB, Attie AD. Overexpression of pre-pro-cholecystokinin stimulates beta-cell proliferation in mouse and human islets with retention of islet function. Molecular Endocrinology (Baltimore, Md.). 22: 2716-28. PMID 18845673 DOI: 10.1210/Me.2008-0255 |
0.756 |
|
| Show low-probability matches. |