Helle D. Ulrich
Affiliations: | Institute of Molecular Biology Mainz, Germany |
Area:
Molecular Biology, Biochemistry, GeneticsGoogle:
"Helle Ulrich"Mean distance: (not calculated yet)
Parents
Sign in to add mentorPeter G. Schultz | grad student | 1992- | UC Berkeley |
Stefan Jentsch | post-doc | Max Planck Institute for Biochemistry |
Children
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Publications
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Son K, Takhaveev V, Mor V, et al. (2024) Trabectedin derails transcription-coupled nucleotide excision repair to induce DNA breaks in highly transcribed genes. Nature Communications. 15: 1388 |
Renz C, Asimaki E, Meister C, et al. (2023) Ubiquiton-An inducible, linkage-specific polyubiquitylation tool. Molecular Cell |
Wollscheid HP, Ulrich HD. (2023) Chromatin meets the cytoskeleton: the importance of nuclear actin dynamics and associated motors for genome stability. Dna Repair. 131: 103571 |
Yakoub G, Choi YS, Wong RP, et al. (2023) Avidity-based biosensors for ubiquitylated PCNA reveal choreography of DNA damage bypass. Science Advances. 9: eadf3041 |
Shi J, Hauschulte K, Mikicic I, et al. (2023) Nuclear myosin VI maintains replication fork stability. Nature Communications. 14: 3787 |
Oravcová M, Nie M, Zilio N, et al. (2022) The Nse5/6-like SIMC1-SLF2 complex localizes SMC5/6 to viral replication centers. Elife. 11 |
Corey DR, Ulrich HD. (2022) Editorial: Critical reviews and perspectives in Nucleic Acids Research. Nucleic Acids Research. 50: 7201 |
Wegmann S, Meister C, Renz C, et al. (2022) Linkage reprogramming by tailor-made E3s reveals polyubiquitin chain requirements in DNA-damage bypass. Molecular Cell |
Asimaki E, Petriukov K, Renz C, et al. (2021) Fast friends - Ubiquitin-like modifiers as engineered fusion partners. Seminars in Cell & Developmental Biology |
Cabello-Lobato MJ, González-Garrido C, Cano-Linares MI, et al. (2021) Physical interactions between MCM and Rad51 facilitate replication fork lesion bypass and ssDNA gap filling by non-recombinogenic functions. Cell Reports. 36: 109440 |