6 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2014 Nielsen N, Pascal V, Fasth AE, Sundström Y, Galsgaard ED, Ahern D, Andersen M, Baslund B, Bartels EM, Bliddal H, Feldmann M, Malmström V, Berg L, Spee P, Söderström K. Balance between activating NKG2D, DNAM-1, NKp44 and NKp46 and inhibitory CD94/NKG2A receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts. Immunology. 142: 581-93. PMID 24673109 DOI: 10.1111/imm.12271  0.31
2008 Li H, Pascal V, Martin MP, Carrington M, Anderson SK. Genetic control of variegated KIR gene expression: polymorphisms of the bi-directional KIR3DL1 promoter are associated with distinct frequencies of gene expression. Plos Genetics. 4: e1000254. PMID 19008943 DOI: 10.1371/journal.pgen.1000254  0.433
2008 Whittaker GC, Burshtyn DN, Orr SJ, Quigley L, Hodge DL, Pascal V, Zhang W, McVicar DW. Analysis of the linker for activation of T cells and the linker for activation of B cells in natural killer cells reveals a novel signaling cassette, dual usage in ITAM signaling, and influence on development of the Ly49 repertoire. Blood. 112: 2869-77. PMID 18645037 DOI: 10.1182/Blood-2007-11-121590  0.464
2007 Pascal V, Yamada E, Martin MP, Alter G, Altfeld M, Metcalf JA, Baseler MW, Adelsberger JW, Carrington M, Anderson SK, McVicar DW. Detection of KIR3DS1 on the cell surface of peripheral blood NK cells facilitates identification of a novel null allele and assessment of KIR3DS1 expression during HIV-1 infection. Journal of Immunology (Baltimore, Md. : 1950). 179: 1625-33. PMID 17641029 DOI: 10.4049/Jimmunol.179.3.1625  0.429
2007 Berthet C, Rodriguez-Galan MC, Hodge DL, Gooya J, Pascal V, Young HA, Keller J, Bosselut R, Kaldis P. Hematopoiesis and thymic apoptosis are not affected by the loss of Cdk2. Molecular and Cellular Biology. 27: 5079-89. PMID 17485443 DOI: 10.1128/Mcb.00029-07  0.307
2004 Pascal V, Schleinitz N, Brunet C, Ravet S, Bonnet E, Lafarge X, Touinssi M, Reviron D, Viallard JF, Moreau JF, Déchanet-Merville J, Blanco P, Harlé JR, Sampol J, Vivier E, et al. Comparative analysis of NK cell subset distribution in normal and lymphoproliferative disease of granular lymphocyte conditions. European Journal of Immunology. 34: 2930-40. PMID 15368309 DOI: 10.1002/eji.200425146  0.31
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