Veronica M. Maher

Affiliations: 
Michigan State University, East Lansing, MI 
Area:
Microbiology Biology
Google:
"Veronica Maher"
BETA: Related publications

Publications

You can help our author matching system! If you notice any publications incorrectly attributed to this author, please sign in and mark matches as correct or incorrect.

Bhat A, Wu Z, Maher VM, et al. (2015) Rev7/Mad2B plays a critical role in the assembly of a functional mitotic spindle. Cell Cycle (Georgetown, Tex.). 14: 3929-38
McCormick JJ, Maher VM. (2011) Malignant transformation of human skin fibroblasts by two alternative pathways. Advances in Experimental Medicine and Biology. 720: 191-207
Neal JA, Fletcher KL, McCormick JJ, et al. (2010) The role of hRev7, the accessory subunit of hPolζ, in translesion synthesis past DNA damage induced by benzo[a]pyrene diol epoxide (BPDE). Bmc Cell Biology. 11: 97
Appledorn DM, Dao KH, O'Reilly S, et al. (2010) Rac1 and Cdc42 are regulators of HRasV12-transformation and angiogenic factors in human fibroblasts. Bmc Cancer. 10: 13
Lito P, Mets BD, Appledorn DM, et al. (2009) Sprouty 2 regulates DNA damage-induced apoptosis in Ras-transformed human fibroblasts. The Journal of Biological Chemistry. 284: 848-54
Li Z, Zhang H, McManus TP, et al. (2009) Erratum to: “hREV3 is essential for error-prone translesion synthesis past UV or benzo[a]pyrene diol epoxide-induced DNA lesions in human fibroblasts” [Mutat. Res. 510 (2002) 71–80] Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 670: 107-108
McNally K, Neal JA, McManus TP, et al. (2008) hRev7, putative subunit of hPolzeta, plays a critical role in survival, induction of mutations, and progression through S-phase, of UV((254nm))-irradiated human fibroblasts. Dna Repair. 7: 597-604
Lito P, Mets BD, Kleff S, et al. (2008) Evidence that sprouty 2 is necessary for sarcoma formation by H-Ras oncogene-transformed human fibroblasts. The Journal of Biological Chemistry. 283: 2002-9
Wang Y, Woodgate R, McManus TP, et al. (2007) Evidence that in xeroderma pigmentosum variant cells, which lack DNA polymerase eta, DNA polymerase iota causes the very high frequency and unique spectrum of UV-induced mutations. Cancer Research. 67: 3018-26
Richards S, Liu ST, Majumdar A, et al. (2005) Triplex targeted genomic crosslinks enter separable deletion and base substitution pathways. Nucleic Acids Research. 33: 5382-93
See more...