Jean M. Pesola, Ph.D.
Affiliations: | 2005 | Harvard University, Cambridge, MA, United States |
Area:
Microbiology BiologyGoogle:
"Jean Pesola"Parents
Sign in to add mentor| Donald M. Coen | grad student | 2005 | Harvard | |
| (Herpes simplex virus gene expression during latency and reactivation.) | ||||
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Publications
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| Deng Y, Lin Y, Chen S, et al. (2024) Neuronal miR-9 promotes HSV-1 epigenetic silencing and latency by repressing Oct-1 and Onecut family genes. Nature Communications. 15: 1991 |
| Chen H, Lye MF, Gorgulla C, et al. (2023) A small molecule exerts selective antiviral activity by targeting the human cytomegalovirus nuclear egress complex. Plos Pathogens. 19: e1011781 |
| Wilkie AR, Sharma M, Coughlin M, et al. (2022) Human Cytomegalovirus Nuclear Egress Complex Subunit, UL53, Associates with Capsids and Myosin Va, but Is Not Important for Capsid Localization towards the Nuclear Periphery. Viruses. 14 |
| Sun B, Yang X, Hou F, et al. (2021) Regulation of host and virus genes by neuronal miR-138 favours herpes simplex virus 1 latency. Nature Microbiology |
| Sen P, Wilkie AR, Ji F, et al. (2020) Linking indirect effects of cytomegalovirus in transplantation to modulation of monocyte innate immune function. Science Advances. 6: eaax9856 |
| Patel CD, Backes IM, Taylor SA, et al. (2019) Maternal immunization confers protection against neonatal herpes simplex mortality and behavioral morbidity. Science Translational Medicine. 11 |
| Lee JS, Raja P, Pan D, et al. (2018) CCCTC-Binding Factor Acts as a Heterochromatin Barrier on Herpes Simplex Viral Latent Chromatin and Contributes to Poised Latent Infection. Mbio. 9 |
| Wilkie AR, Sharma M, Pesola JM, et al. (2018) A role for myosin Va in human cytomegalovirus nuclear egress. Journal of Virology |
| Pan D, Pesola JM, Li G, et al. (2016) Mutations Inactivating Herpes Simplex Virus-1 miR-H2 Do Not Detectably Increase ICP0 Gene Expression in Infected Cultured Cells or Mouse Trigeminal Ganglia. Journal of Virology |
| Rosato PC, Katzenell S, Pesola JM, et al. (2016) Neuronal IFN signaling is dispensable for the establishment of HSV-1 latency. Virology. 497: 323-327 |