Srikanta Dash
Affiliations: | Biomedical Sciences | Tulane University, New Orleans, LA, United States |
Area:
Virology Biology, Molecular Biology, Microbiology BiologyGoogle:
"Srikanta Dash"
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Publications
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| Chandra PK, Kundu AK, Hazari S, et al. (2024) Retraction Notice to: Inhibition of Hepatitis C Virus Replication by Intracellular Delivery of Multiple siRNAs by Nanosomes. Molecular Therapy : the Journal of the American Society of Gene Therapy |
| Chandra PK, Hazari S, Poat B, et al. (2024) Correction: Intracytoplasmic stable expression of IgG1 antibody targeting NS3 helicase inhibits replication of highly efficient hepatitis C Virus 2a clone. Virology Journal. 21: 274 |
| Lin D, Chen Y, Koksal AR, et al. (2023) Targeting ER stress/PKA/GSK-3β/β-catenin pathway as a potential novel strategy for hepatitis C virus-infected patients. Cell Communication and Signaling : Ccs. 21: 102 |
| Aydin Y, Chedid M, Chava S, et al. (2021) Retraction Note: Activation of PERK-Nrf2 oncogenic signaling promotes Mdm2-mediated Rb degradation in persistently infected HCV culture. Scientific Reports. 11: 17779 |
| Prabhu R, Garry RF, Dash S. (2021) Retraction Note: Small interfering RNA targeted to stem-loop II of the 5' untranslated region effectively inhibits expression of six HCV genotypes. Virology Journal. 18: 155 |
| Aydin Y, Koksal AR, Reddy V, et al. (2021) Extracellular Vesicle Release Promotes Viral Replication during Persistent HCV Infection. Cells. 10 |
| Lin D, Reddy V, Osman H, et al. (2021) Additional Inhibition of Wnt/β-Catenin Signaling by Metformin in DAA Treatments as a Novel Therapeutic Strategy for HCV-Infected Patients. Cells. 10 |
| Dash S, Aydin Y, Widmer KE, et al. (2020) Hepatocellular Carcinoma Mechanisms Associated with Chronic HCV Infection and the Impact of Direct-Acting Antiviral Treatment. Journal of Hepatocellular Carcinoma. 7: 45-76 |
| Dash S, Aydin Y, Moroz K. (2019) Chaperone-Mediated Autophagy in the Liver: Good or Bad? Cells. 8 |
| Song K, Kwon H, Han C, et al. (2019) YAP in Kupffer cells enhances the production of pro-inflammatory cytokines and promotes the development of non-alcoholic steatohepatitis. Hepatology (Baltimore, Md.) |