Ching W. Sze, Ph.D.
Affiliations: | 2012 | Oral Biology | State University of New York, Buffalo, Buffalo, NY, United States |
Area:
Microbiology BiologyGoogle:
"Ching Sze"Parents
Sign in to add mentor| Chunhao ( Li | grad student | 2012 | SUNY Buffalo | |
| (Regulation of chemotaxis, motility, and virulence in the lyme disease spirochete Borrelia burgdorferi.) | ||||
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Publications
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| Sze CW, Zhang K, Lynch MJ, et al. (2025) The Flagellin-Specific Chaperone FliS of Borrelia burgdorferi Controls the Cytoplasmic Pool of Flagellins at the Level of Translation Initiation, Secretion, and Proteolysis. Molecular Microbiology |
| Sze CW, Lynch MJ, Zhang K, et al. (2025) Lactate dehydrogenase is the Achilles' heel of Lyme disease bacterium . Mbio. e0372824 |
| Zhang K, Sze CW, Zhao H, et al. (2025) Borrelia burgdorferi serine protease HtrA is a pleiotropic regulator of stress response, motility, flagellar hemostasis, and infectivity. Communications Biology. 8: 341 |
| Sze CW, Lynch MJ, Zhang K, et al. (2025) Lactate dehydrogenase is the Achilles' heel of Lyme disease bacterium . Biorxiv : the Preprint Server For Biology |
| Kurniyati K, Clark ND, Wang H, et al. (2025) A bipartite bacterial virulence factor targets the complement system and neutrophil activation. The Embo Journal |
| Raghunandanan S, Zhang K, Zhang Y, et al. (2024) MCP5, a methyl-accepting chemotaxis protein regulated by both the Hk1-Rrp1 and Rrp2-RpoN-RpoS pathways, is required for the immune evasion of Borrelia burgdorferi. Plos Pathogens. 20: e1012327 |
| Raghunandanan S, Zhang K, Yan Z, et al. (2024) MCP5, a methyl-accepting chemotaxis protein regulated by both the Hk1-Rrp1 and Rrp2-RpoN-RpoS pathways, is required for the immune evasion of . Biorxiv : the Preprint Server For Biology |
| Pham C, Guo S, Han X, et al. (2024) A pleiotropic role of sialidase in the pathogenicity of . Infection and Immunity. e0034423 |
| Sze CW, Zhang K, Lynch MJ, et al. (2023) A chemosensory-like histidine kinase is dispensable for chemotaxis in vitro but regulates the virulence of Borrelia burgdorferi through modulating the stability of RpoS. Plos Pathogens. 19: e1011752 |
| Clark ND, Pham C, Kurniyati K, et al. (2023) Functional and structural analyses reveal that a dual domain sialidase protects bacteria from complement killing through desialylation of complement factors. Plos Pathogens. 19: e1011674 |