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| Wu J, Zhang L, Shi J, et al. (2020) Macrophage phenotypic switch orchestrates the inflammation and repair/regeneration following acute pancreatitis injury. Ebiomedicine. 58: 102920 |
| Niu N, Lu P, Yang Y, et al. (2019) Loss of Setd2 promotes Kras-induced acinar-to-ductal metaplasia and epithelia-mesenchymal transition during pancreatic carcinogenesis. Gut |
| Shi J, Xue J. (2019) Inflammation and development of pancreatic ductal adenocarcinoma. Chinese Clinical Oncology. 8: 19 |
| Zhang R, Shi J, Zhang R, et al. (2019) Expanded CD14CD16 Immunosuppressive Monocytes Predict the Severity of Patients with Acute Pancreatitis. Journal of Immunology (Baltimore, Md. : 1950) |
| He W, Wu J, Shi J, et al. (2018) IL-22RA1/ STAT3 signaling promotes stemness and tumorigenicity in pancreatic cancer. Cancer Research |
| Xue J, Zhao Q, Sharma V, et al. (2016) Aaryl Hydrocarbon Receptor Ligands in Cigarette Smoke Induce Production of Interleukin-22 to Promote Pancreatic Fibrosis in Models of Chronic Pancreatitis. Gastroenterology |
| Edderkaoui M, Xu S, Chheda C, et al. (2016) HDAC3 mediates smoking-induced pancreatic cancer. Oncotarget |
| Xue J, Zhao Q, Sharma V, et al. (2016) Tu1499 Efficacy of a Small Molecule CCR2 Antagonist in Experimental Model of Chronic Pancreatitis Gastroenterology. 150: S917 |
| Xue J, Sharma V, Park W, et al. (2016) AhR ligands in cigarette smoke mediate immune activation and promote fibrosis in chronic pancreatitis Pancreatology. 16: S40 |
| Xue J, Sharma V, Hsieh MH, et al. (2015) Alternatively activated macrophages promote pancreatic fibrosis in chronic pancreatitis. Nature Communications. 6: 7158 |