Gina M. Ney, Ph.D.
Affiliations: | 2007 | University of Michigan, Ann Arbor, Ann Arbor, MI |
Area:
Medicinal ChemistryGoogle:
"Gina Ney"Parents
Sign in to add mentor| William R. Roush | grad student | 2007 | University of Michigan | |
| (Moelcular recognition of small molecules by the mitochondrial F1F0-ATPase.) | ||||
| Qing Li | post-doc | University of Michigan (Neurotree) | ||
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Publications
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| Jin X, Ng V, Zhao M, et al. (2022) Epigenetic Downregulation of Socs2 Contributes to Mutant N-Ras-Mediated Hematopoietic Dysregulation. Disease Models & Mechanisms |
| Ney GM, Yang KB, Ng V, et al. (2021) Oncogenic N-Ras mitigates oxidative stress-induced apoptosis of hematopoietic stem cells. Cancer Research |
| Liu L, Inoki A, Fan K, et al. (2020) ER associated degradation preserves hematopoietic stem cell quiescence and self-renewal by restricting mTOR activity. Blood |
| Ney GM, McKay LM, Koschmann C, et al. (2020) The Emerging Role of Ras Pathway Signaling in Pediatric Cancer. Cancer Research |
| Ney GM, Anderson B, Bender J, et al. (2020) Mutations predictive of hyperactive Ras signaling correlate with inferior survival across high-risk pediatric acute leukemia. Translational Pediatrics. 9: 43-50 |
| Liu L, Zhao M, Jin X, et al. (2019) Adaptive endoplasmic reticulum stress signalling via IRE1α-XBP1 preserves self-renewal of haematopoietic and pre-leukaemic stem cells. Nature Cell Biology |
| Jin X, Qin T, Zhao M, et al. (2018) Oncogenic N-Ras and Tet2 haploinsufficiency collaborate to dysregulate hematopoietic stem and progenitor cells. Blood Advances. 2: 1259-1271 |
| Liu L, Zhao M, Yang KB, et al. (2016) N-RasG12D-Mediated Dysregulation of IRE1alpha-Xbp1s Signaling Promotes Pre-Leukemic Hematopoietic Stem Cell Expansion Blood. 128: 567-567 |
| Jin X, Qin T, Bailey NG, et al. (2016) Hyperactive Nras and Dose Reduction of Tet2 Collaborate to Dys-Regulate Hematopoietic Stem Cells and Promote Leukemogenesis Blood. 128: 1204-1204 |
| Palmbos PL, Wang L, Yang H, et al. (2015) ATDC (TRIM29) Drives Invasive Bladder Cancer Formation. Cancer Research |