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Susan Michaelis, Ph.D.

Affiliations: 
Cell Biology Johns Hopkins University, Baltimore, MD 
Area:
s.cerevisiae, membrane biology, ABC transporters, protein degradation mechanisms
Website:
http://cellbio.jhmi.edu/people/faculty/susan-michaelis-phd
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"Susan Michaelis"
Bio:

https://scholar.google.com/citations?hl=en&user=CMrWvzUAAAAJ

Mean distance: 13358.2 (cluster 53)
 

Parents

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Jonathan R. Beckwith grad student 1982 Harvard Medical School (Chemistry Tree)
 (Use of gene fusions to obtain mutations which alter the signal sequence of alkaline phosphatase in Escherichia coli)
Ira Herskowitz post-doc UCSF (Chemistry Tree)

Children

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Nisha Susanna Pulimood research assistant Johns Hopkins Medical School
Allen T Yu research assistant 2008-2012 Johns Hopkins Medical School (Cell Biology Tree)
Wenxin W. Xu research assistant 2018-2018 Johns Hopkins
Carol Berkower grad student Johns Hopkins
Julia D. Romano grad student 2000 Johns Hopkins
Amy Tam grad student 2000 Johns Hopkins
Deborah L. Mason grad student 2002 Johns Hopkins
Kellie B. Kelm grad student 2003 Johns Hopkins
Meredith B. Metzger grad student 2009 Johns Hopkins
Matthew J. Maurer grad student 2012 Johns Hopkins
Megan S. Kane grad student 2013 Johns Hopkins
Walter K. Schmidt post-doc 2001 Johns Hopkins Medical School
Christian M. Paumi post-doc 2004-2008 Johns Hopkins Medical School
BETA: Related publications

Publications

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Wang W, Lu J, Yang WC, et al. (2022) Analysis of a degron-containing reporter protein GFP-CL1 reveals a role for SUMO1 in cytosolic protein quality control. The Journal of Biological Chemistry. 102851
Wood KM, Spear ED, Mossberg OW, et al. (2020) Defining substrate requirements for cleavage of farnesylated prelamin A by the integral membrane zinc metalloprotease ZMPSTE24. Plos One. 15: e0239269
Babatz TD, Spear ED, Xu W, et al. (2020) Site specificity determinants for prelamin A cleavage by the zinc metalloprotease ZMPSTE24. The Journal of Biological Chemistry
Spear ED, Alford RF, Babatz TD, et al. (2019) A Humanized Yeast System to Analyze Cleavage of Prelamin A by ZMPSTE24. Methods (San Diego, Calif.)
Spear ED, Hsu ET, Nie L, et al. (2018) missense mutations that cause progeroid diseases decrease prelamin A cleavage activity and/or protein stability. Disease Models & Mechanisms
Preston GM, Guerriero CJ, Metzger MB, et al. (2018) Substrate Insolubility Dictates Hsp104-Dependent Endoplasmic-Reticulum-Associated Degradation. Molecular Cell. 70: 242-253.e6
Maurer MJ, Spear ED, Yu AT, et al. (2016) Degradation Signals for Ubiquitin-Proteasome Dependent Cytosolic Protein Quality Control (CytoQC) in Yeast. G3 (Bethesda, Md.)
Ast T, Michaelis S, Schuldiner M. (2016) The Protease Ste24 Clears Clogged Translocons. Cell. 164: 103-14
Snider J, Hanif A, Lee ME, et al. (2013) Mapping the functional yeast ABC transporter interactome. Nature Chemical Biology. 9: 565-72
Kane MS, Lindsay ME, Judge DP, et al. (2013) LMNA-associated cardiocutaneous progeria: an inherited autosomal dominant premature aging syndrome with late onset. American Journal of Medical Genetics. Part A. 161: 1599-611
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