Michael K. Lee, PhD
Affiliations: | Pathology | Johns Hopkins University, Baltimore, MD | |
| Institute for Translational Neuroscience | University of Minnesota, Twin Cities, Minneapolis, MN |
Area:
pathogenic mechanisms occurring in human neurodegenerative diseasesWebsite:
https://neurosci.umn.edu/bio/department-of-neuroscience/michael-leeGoogle:
"Michael Lee"Bio:
http://pds17.pathology.jhmi.edu/N/n.web?EP=N&URL=/MCGI/SEND^WEBUTLTY(1058)/1543522402
Mean distance: 53433
Collaborators
Sign in to add collaborator| Dezhi Liao | collaborator | UMN | |
| Dalila G. Ordonez | collaborator |
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Publications
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| Vermilyea SC, Christensen A, Meints J, et al. (2022) Loss of tau expression attenuates neurodegeneration associated with α-synucleinopathy. Translational Neurodegeneration. 11: 34 |
| Karim MR, Liao EE, Kim J, et al. (2020) α-Synucleinopathy associated c-Abl activation causes p53-dependent autophagy impairment. Molecular Neurodegeneration. 15: 27 |
| Stanojlovic M, Pallais JP, Lee MK, et al. (2019) Pharmacological and chemogenetic orexin/hypocretin intervention ameliorates Hipp-dependent memory impairment in the A53T mice model of Parkinson's disease. Molecular Brain. 12: 87 |
| Singh B, Covelo A, Martell-Martínez H, et al. (2019) Tau is required for progressive synaptic and memory deficits in a transgenic mouse model of α-synucleinopathy. Acta Neuropathologica |
| Teravskis PJ, Covelo A, Miller EC, et al. (2018) A53T mutant alpha-synuclein induces tau dependent postsynaptic impairment independent of neurodegenerative changes. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Khan SS, LaCroix M, Boyle G, et al. (2018) Bidirectional modulation of Alzheimer phenotype by alpha-synuclein in mice and primary neurons. Acta Neuropathologica |
| Ordonez DG, Lee MK, Feany MB. (2017) α-synuclein Induces Mitochondrial Dysfunction through Spectrin and the Actin Cytoskeleton. Neuron |
| Colla E, Panattoni G, Ricci A, et al. (2017) Toxic properties of microsome-associated alpha-synuclein species in mouse primary neurons. Neurobiology of Disease. 111: 36-47 |
| Salazar SV, Gallardo C, Kaufman AC, et al. (2017) Conditional Deletion of Prnp Rescues Behavioral and Synaptic Deficits after Disease Onset in Transgenic Alzheimer's Disease. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience |
| Larson ME, Greimel SJ, Amar F, et al. (2017) Selective lowering of synapsins induced by oligomeric α-synuclein exacerbates memory deficits. Proceedings of the National Academy of Sciences of the United States of America |