Vivian J. Bardwell
Affiliations: | University of Minnesota, Twin Cities, Minneapolis, MN |
Area:
Molecular Biology, Genetics, Cell BiologyGoogle:
"Vivian Bardwell"Children
Sign in to add trainee| Khanh D. Huynh | grad student | 2000 | UMN |
| Umut Fahrioglu | grad student | 2004 | UMN |
| Joseph A. Wamstad | grad student | 2009 | UMN |
| Michelle Y. Hamline | grad student | 2011 | UMN |
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Publications
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| Sadowski D, Corcoran CM, Abdi R, et al. (2024) Polycomb Repressive Complex 1.1 Component, BCOR, Promotes Syncytiotrophoblast Differentiation in Mice and Humans. Biorxiv : the Preprint Server For Biology |
| Murphy MW, Gearhart MD, Wheeler A, et al. (2022) Genomics of sexual cell fate transdifferentiation in the mouse gonad. G3 (Bethesda, Md.) |
| Langouët M, Jolicoeur C, Javed A, et al. (2022) Mutations in , a co-repressor of , are associated with early-onset retinal degeneration. Science Advances. 8: eabh2868 |
| Agrimson KS, Minkina A, Sadowski D, et al. (2022) Lrh1 can help reprogram sexual cell fate and is required for Sertoli cell development and spermatogenesis in the mouse testis. Plos Genetics. 18: e1010088 |
| Giefing M, Gearhart MD, Schneider M, et al. (2021) Loss of function mutations of in classical Hodgkin lymphoma. Leukemia & Lymphoma. 1-11 |
| Lindeman RE, Murphy MW, Agrimson KS, et al. (2021) The conserved sex regulator DMRT1 recruits SOX9 in sexual cell fate reprogramming. Nucleic Acids Research |
| Kutscher LM, Okonechnikov K, Batora NV, et al. (2020) Functional loss of a noncanonical BCOR-PRC1.1 complex accelerates SHH-driven medulloblastoma formation. Genes & Development |
| Hamline MY, Corcoran CM, Wamstad JA, et al. (2020) OFCD syndrome and extraembryonic defects are revealed by conditional mutation of the polycomb-group repressive complex 1.1 (PRC1.1) gene BCOR. Developmental Biology |
| Wong SJ, Senkovich O, Artigas JA, et al. (2020) Structure and role of BCOR PUFD in non-canonical PRC1 assembly and disease. Biochemistry |
| Kotov JA, Kotov DI, Linehan JL, et al. (2019) BCL6 corepressor contributes to Th17 cell formation by inhibiting Th17 fate suppressors. The Journal of Experimental Medicine |