Joseph R. Mazzulli, Ph.D.

Affiliations: 
2007 University of Pennsylvania, Philadelphia, PA, United States 
Area:
Neuroscience Biology
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Cross-listing: Neurotree

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Harry Ischiropoulos grad student 2007 Penn
 (Catechol oxidation and alpha-synuclein aggregation: Implications for selective dopaminergic neurodegeneration in Parkinson's disease.)
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Publications

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Shtilbans A, Esneault E, Simon F, et al. (2025) Evaluation of Additive Neuroprotective Effect of Combination Therapy for Parkinson's Disease Using In Vitro Models. Antioxidants (Basel, Switzerland). 14
Sokratian A, Zhou Y, Tatli M, et al. (2024) Mouse α-synuclein fibrils are structurally and functionally distinct from human fibrils associated with Lewy body diseases. Science Advances. 10: eadq3539
Belur NR, Bustos BI, Lubbe SJ, et al. (2024) Nuclear aggregates of NONO/SFPQ and A-to-I-edited RNA in Parkinson's disease and dementia with Lewy bodies. Neuron
Zalon AJ, Quiriconi DJ, Pitcairn C, et al. (2024) α-Synuclein: Multiple pathogenic roles in trafficking and proteostasis pathways in Parkinson's disease. The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry. 10738584241232963
Fagen SJ, Burgess JD, Lim MJ, et al. (2023) Honokiol decreases alpha-synuclein mRNA levels and reveals novel targets for modulating alpha-synuclein expression. Frontiers in Aging Neuroscience. 15: 1179086
Drobny A, Boros FA, Balta D, et al. (2023) Reciprocal effects of alpha-synuclein aggregation and lysosomal homeostasis in synucleinopathy models. Translational Neurodegeneration. 12: 31
Pitcairn C, Murata N, Zalon A, et al. (2023) Impaired autophagic-lysosomal fusion in Parkinson's patient midbrain neurons occurs through loss of ykt6 and is rescued by farnesyltransferase inhibition. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience
Prieto Huarcaya S, Drobny A, Marques ARA, et al. (2022) Recombinant pro-CTSD (cathepsin D) enhances SNCA/α-Synuclein degradation in α-Synucleinopathy models. Autophagy
Francelle L, Mazzulli JR. (2022) Neuroinflammation in Gaucher disease, neuronal ceroid lipofuscinosis, and commonalities with Parkinson's disease. Brain Research. 1780: 147798
Fredriksen K, Aivazidis S, Sharma K, et al. (2021) Pathological α-syn aggregation is mediated by glycosphingolipid chain length and the physiological state of α-syn in vivo. Proceedings of the National Academy of Sciences of the United States of America. 118
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