Patrick C. Bradshaw, PhD
Affiliations: | 2015- | Department of Biomedical Sciences | East Tennessee State University - Quillen School of Medicine |
Area:
mitochondrial function, aging, neurodegenerationWebsite:
http://www.etsu.edu/com/dbms/faculty/bios/bradshaw.aspxGoogle:
"Patrick Bradshaw"Bio:
Dr. Bradshaw grew up in East Peoria, IL, attended the University of Illinois for his B.S. degree, majoring in bioengineering and chemistry. He went on to earn a PhD in biochemistry at Ohio State University followed by postdoctoral research at the University of Missouri-Columbia, Virginia Tech, and the University of Wisconsin Madison. He was a faculty member at the University of South Florida from 2008-2015.
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Parents
Sign in to add mentor| Douglas R. Pfeiffer | grad student | 1995-2001 | Ohio State University Medical Center |
| David C. Samuels | post-doc | 2003-2005 | Vanderbilt University Medical School |
| Tomas A. Prolla | post-doc | 2005-2007 | UW Madison (Cell Biology Tree) |
| Richard Weindruch | post-doc | 2005-2007 | UW Madison |
Children
Sign in to add trainee| Jeddidiah WD Griffin | grad student | 2016- | East Tennessee State University - Quillen School of Medicine |
| Neil Copes | grad student | 2009-2015 | University of South Florida |
| Vedad Delic | grad student | 2011-2015 | University of South Florida |
| Clare Edwards-Canfield | grad student | 2011-2015 | University of South Florida |
BETA: Related publications
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Publications
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| Bradshaw PC, Aldridge JL, Jamerson LE, et al. (2024) The Role of Cardiolipin in Brain Bioenergetics, Neuroinflammation, and Neurodegeneration. Molecular Neurobiology |
| Curtis WM, Seeds WA, Mattson MP, et al. (2022) NADPH and Mitochondrial Quality Control as Targets for a Circadian-Based Fasting and Exercise Therapy for the Treatment of Parkinson's Disease. Cells. 11 |
| Bradshaw PC. (2019) Cytoplasmic and Mitochondrial NADPH-Coupled Redox Systems in the Regulation of Aging. Nutrients. 11 |
| Delic V, Noble K, Zivkovic S, et al. (2018) The effects of AICAR and rapamycin on mitochondrial function in immortalized mitochondrial DNA mutator murine embryonic fibroblasts. Biology Open |
| Delic V, Kurien C, Cruz J, et al. (2018) Discrete mitochondrial aberrations in the spinal cord of sporadic ALS patients. Journal of Neuroscience Research |
| Delic V, Griffin JWD, Zivkovic S, et al. (2017) Individual Amino Acid Supplementation Can Improve Energy Metabolism and Decrease ROS Production in Neuronal Cells Overexpressing Alpha-Synuclein. Neuromolecular Medicine |
| Sawmiller D, Li S, Mori T, et al. (2017) Beneficial effects of a pyrroloquinolinequinone-containing dietary formulation on motor deficiency, cognitive decline and mitochondrial dysfunction in a mouse model of Alzheimer's disease. Heliyon. 3: e00279 |
| Griffin JW, Bradshaw PC. (2017) Amino Acid Catabolism in Alzheimer's Disease Brain: Friend or Foe? Oxidative Medicine and Cellular Longevity. 2017: 5472792 |
| Copes N, Edwards C, Chaput D, et al. (2015) Metabolome and proteome changes with aging in Caenorhabditis elegans. Experimental Gerontology. 72: 67-84 |
| O'Neal-Moffitt G, Delic V, Bradshaw PC, et al. (2015) Prophylactic melatonin significantly reduces Alzheimer's neuropathology and associated cognitive deficits independent of antioxidant pathways in AβPP(swe)/PS1 mice. Molecular Neurodegeneration. 10: 27 |