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Y Mishina - Publications

Affiliations: 
2006-2009 Pediatrics and Physiology University of Chicago, Chicago, IL 
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7 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2020 Hu Y, Hao X, Liu C, Ren C, Wang S, Yan G, Meng Y, Mishina Y, Shi C, Sun H. Acvr1 deletion in osteoblasts impaired mandibular bone mass through compromised osteoblast differentiation and enhanced sRANKL-induced osteoclastogenesis. Journal of Cellular Physiology. PMID 33251612 DOI: 10.1002/jcp.30183  0.303
2020 Kamiya N, Atsawasuwan P, Joiner DM, Waldorff EI, Goldstein S, Yamauchi M, Mishina Y. Controversy of physiological vs. pharmacological effects of BMP signaling: Constitutive activation of BMP type IA receptor-dependent signaling in osteoblast lineage enhances bone formation and resorption, not affecting net bone mass. Bone. 115513. PMID 32603910 DOI: 10.1016/J.Bone.2020.115513  0.562
2020 Zhang H, Zhang Y, Terajima M, Romanowicz G, Liu Y, Omi M, Bigelow E, Joiner DM, Waldorff EI, Zhu P, Raghavan M, Lynch M, Kamiya N, Zhang R, Jepsen KJ, ... ... Mishina Y, et al. Loss of BMP signaling mediated by BMPR1A in osteoblasts leads to differential bone phenotypes in mice depending on anatomical location of the bones. Bone. 115402. PMID 32360900 DOI: 10.1016/J.Bone.2020.115402  0.55
2018 Biswas S, Li P, Wu H, Shafiquzzaman M, Murakami S, Schneider MD, Mishina Y, Li B, Li J. BMPRIA is required for osteogenic differentiation and RANKL expression in adult bone marrow mesenchymal stromal cells. Scientific Reports. 8: 8475. PMID 29855498 DOI: 10.1038/S41598-018-26820-8  0.302
2016 Shi C, Iura A, Terajima M, Liu F, Lyons K, Pan H, Zhang H, Yamauchi M, Mishina Y, Sun H. Deletion of BMP receptor type IB decreased bone mass in association with compromised osteoblastic differentiation of bone marrow mesenchymal progenitors. Scientific Reports. 6: 24256. PMID 27048979 DOI: 10.1038/Srep24256  0.307
2013 Ruscic KJ, Miceli F, Villalba-Galea CA, Dai H, Mishina Y, Bezanilla F, Goldstein SA. IKs channels open slowly because KCNE1 accessory subunits slow the movement of S4 voltage sensors in KCNQ1 pore-forming subunits. Proceedings of the National Academy of Sciences of the United States of America. 110: E559-66. PMID 23359697 DOI: 10.1073/Pnas.1222616110  0.416
2011 Kamiya N, Kaartinen VM, Mishina Y. Loss-of-function of ACVR1 in osteoblasts increases bone mass and activates canonical Wnt signaling through suppression of Wnt inhibitors SOST and DKK1. Biochemical and Biophysical Research Communications. 414: 326-30. PMID 21945937 DOI: 10.1016/J.Bbrc.2011.09.060  0.315
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