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Qi Li - Publications

Affiliations: 
Wuhan University, Wuhan Shi, Hubei Sheng, China 
 2012- Cell Biology Hospital for Sick Children, Toronto, ON 
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10 high-probability publications. We are testing a new system for linking publications to authors. You can help! If you notice any inaccuracies, please sign in and mark papers as correct or incorrect matches. If you identify any major omissions or other inaccuracies in the publication list, please let us know.

Year Citation  Score
2022 Wang L, Aschenbrenner D, Zeng Z, Cao X, Mayr D, Mehta M, Capitani M, Warner N, Pan J, Wang L, Li Q, Zuo T, Cohen-Kedar S, Lu J, Ardy RC, et al. Author Correction: Gain-of-function variants in SYK cause immune dysregulation and systemic inflammation in humans and mice. Nature Genetics. PMID 34987219 DOI: 10.1038/s41588-021-00998-6  0.52
2021 Wang L, Aschenbrenner D, Zeng Z, Cao X, Mayr D, Mehta M, Capitani M, Warner N, Pan J, Wang L, Li Q, Zuo T, Cohen-Kedar S, Lu J, Ardy RC, et al. Gain-of-function variants in SYK cause immune dysregulation and systemic inflammation in humans and mice. Nature Genetics. PMID 33782605 DOI: 10.1038/s41588-021-00803-4  0.574
2021 Bosa L, Batura V, Colavito D, Fiedler K, Gaio P, Guo C, Li Q, Marzollo A, Mescoli C, Nambu R, Pan J, Perilongo G, Warner N, Zhang S, Kotlarz D, et al. Novel CARMIL2 loss-of-function variants are associated with pediatric inflammatory bowel disease. Scientific Reports. 11: 5945. PMID 33723309 DOI: 10.1038/s41598-021-85399-9  0.58
2020 Dhingani N, Guo C, Pan J, Li Q, Warner N, Jardine S, Leung G, Kotlarz D, Gonzaga-Jauregui C, Klein C, Snapper SB, Navas-López VM, Muise AM. The E3 ubiquitin ligase UBR5 interacts with TTC7A and may be associated with very early onset inflammatory bowel disease. Scientific Reports. 10: 18648. PMID 33122718 DOI: 10.1038/s41598-020-73482-6  0.571
2017 Kahr WH, Pluthero FG, Elkadri A, Warner N, Drobac M, Chen CH, Lo RW, Li L, Li R, Li Q, Thoeni C, Pan J, Leung G, Lara-Corrales I, Murchie R, et al. Loss of the Arp2/3 complex component ARPC1B causes platelet abnormalities and predisposes to inflammatory disease. Nature Communications. 8: 14816. PMID 28368018 DOI: 10.1038/Ncomms14816  0.61
2016 Li Q, Lee CH, Peters LA, Mastropaolo LA, Thoeni C, Elkadri A, Schwerd T, Zhu J, Zhang B, Zhao Y, Hao K, Dinarzo A, Hoffman G, Kidd BA, Murchie R, et al. Variants in TRIM22 that Affect NOD2 Signaling Are Associated With Very Early Onset Inflammatory Bowel Disease. Gastroenterology. PMID 26836588 DOI: 10.1053/J.Gastro.2016.01.031  0.619
2014 Yan J, Li Q, Mao AP, Hu MM, Shu HB. TRIM4 modulates type I interferon induction and cellular antiviral response by targeting RIG-I for K63-linked ubiquitination. Journal of Molecular Cell Biology. 6: 154-63. PMID 24755855 DOI: 10.1093/jmcb/mju005  0.495
2013 Zheng H, Li Q, Chen R, Zhang J, Ran Y, He X, Li S, Shu HB. The dual-specificity phosphatase DUSP14 negatively regulates tumor necrosis factor- and interleukin-1-induced nuclear factor-κB activation by dephosphorylating the protein kinase TAK1. The Journal of Biological Chemistry. 288: 819-25. PMID 23229544 DOI: 10.1074/Jbc.M112.412643  0.491
2011 Li Q, Yan J, Mao AP, Li C, Ran Y, Shu HB, Wang YY. Tripartite motif 8 (TRIM8) modulates TNFα- and IL-1β-triggered NF-κB activation by targeting TAK1 for K63-linked polyubiquitination. Proceedings of the National Academy of Sciences of the United States of America. 108: 19341-6. PMID 22084099 DOI: 10.1073/pnas.1110946108  0.482
2010 Mao AP, Li S, Zhong B, Li Y, Yan J, Li Q, Teng C, Shu HB. Virus-triggered ubiquitination of TRAF3/6 by cIAP1/2 is essential for induction of interferon-beta (IFN-beta) and cellular antiviral response. The Journal of Biological Chemistry. 285: 9470-6. PMID 20097753 DOI: 10.1074/jbc.M109.071043  0.478
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