Year |
Citation |
Score |
2012 |
Raake PW, Zhang X, Vinge LE, Brinks H, Gao E, Jaleel N, Li Y, Tang M, Most P, Dorn GW, Houser SR, Katus HA, Chen X, Koch WJ. Cardiac G-protein-coupled receptor kinase 2 ablation induces a novel Ca2+ handling phenotype resistant to adverse alterations and remodeling after myocardial infarction. Circulation. 125: 2108-18. PMID 22496128 DOI: 10.1161/Circulationaha.111.044255 |
0.729 |
|
2010 |
Zhang H, Chen X, Gao E, MacDonnell SM, Wang W, Kolpakov M, Nakayama H, Zhang X, Jaleel N, Harris DM, Li Y, Tang M, Berretta R, Leri A, Kajstura J, et al. Increasing cardiac contractility after myocardial infarction exacerbates cardiac injury and pump dysfunction Circulation Research. 107: 800-809. PMID 20671241 DOI: 10.1016/J.Bpj.2008.12.1276 |
0.759 |
|
2009 |
Kubo H, Berretta RM, Jaleel N, Angert D, Houser SR. c-Kit+ bone marrow stem cells differentiate into functional cardiac myocytes. Clinical and Translational Science. 2: 26-32. PMID 20443864 DOI: 10.1111/J.1752-8062.2008.00089.X |
0.677 |
|
2009 |
MacDonnell SM, Weisser-Thomas J, Kubo H, Hanscome M, Liu Q, Jaleel N, Berretta R, Chen X, Brown JH, Sabri AK, Molkentin JD, Houser SR. CaMKII negatively regulates calcineurin-NFAT signaling in cardiac myocytes. Circulation Research. 105: 316-25. PMID 19608982 DOI: 10.1161/Circresaha.109.194035 |
0.73 |
|
2008 |
Jaleel N, Nakayama H, Chen X, Kubo H, MacDonnell S, Zhang H, Berretta R, Robbins J, Cribbs L, Molkentin JD, Houser SR. Ca2+ influx through T- and L-type Ca2+ channels have different effects on myocyte contractility and induce unique cardiac phenotypes. Circulation Research. 103: 1109-19. PMID 18832749 DOI: 10.1161/Circresaha.108.185611 |
0.748 |
|
2008 |
Kubo H, Jaleel N, Kumarapeli A, Berretta RM, Bratinov G, Shan X, Wang H, Houser SR, Margulies KB. Increased cardiac myocyte progenitors in failing human hearts. Circulation. 118: 649-57. PMID 18645055 DOI: 10.1016/J.Cardfail.2008.06.102 |
0.689 |
|
2007 |
Nakayama H, Chen X, Baines CP, Klevitsky R, Zhang X, Zhang H, Jaleel N, Chua BH, Hewett TE, Robbins J, Houser SR, Molkentin JD. Ca2+- and mitochondrial-dependent cardiomyocyte necrosis as a primary mediator of heart failure. The Journal of Clinical Investigation. 117: 2431-44. PMID 17694179 DOI: 10.1172/Jci31060 |
0.71 |
|
2007 |
Chen X, Wilson RM, Kubo H, Berretta RM, Harris DM, Zhang X, Jaleel N, MacDonnell SM, Bearzi C, Tillmanns J, Trofimova I, Hosoda T, Mosna F, Cribbs L, Leri A, et al. Adolescent feline heart contains a population of small, proliferative ventricular myocytes with immature physiological properties. Circulation Research. 100: 536-44. PMID 17272809 DOI: 10.1161/01.Res.0000259560.39234.99 |
0.733 |
|
2007 |
Jaleel N, Kubo H, Macdonnell S, Chen X, Berretta R, Zhang H, Angert D, Brown JH, Houser SR. Constitutive Activation of CaMKII Reduces Cardiac Function and Viability in Feline Ventricular Myocytes Journal of Cardiac Failure. 13: S94-S95. DOI: 10.1016/J.Cardfail.2007.06.373 |
0.653 |
|
2007 |
Zhang H, Chen X, Gao E, Harris D, Nakayama H, Zhang X, Jaleel N, Eckhart A, Koch W, Molkentin J, Houser S. Excess Ca2+ Influx through the L-Type Ca2+ Channel Exacerbates Ischemia-Induced Cardiac Damage Journal of Cardiac Failure. 13: S87-S88. DOI: 10.1016/J.Cardfail.2007.06.348 |
0.72 |
|
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