Tzong-Jen Sheu, Ph.D.
Affiliations: | 2002 | University of Rochester, Rochester, NY |
Area:
Pathology, Animal Physiology BiologyGoogle:
"Tzong-Jen Sheu"Parents
Sign in to add mentor| J Edward Puzas | grad student | 2002 | Rochester | |
| (Site -specific regulation of osteoblast activity.) | ||||
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Publications
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| Tsai CH, Chang CY, Lin BZ, et al. (2020) Up-regulation of cofilin-1 in cell senescence associates with morphological change and p27 -mediated growth delay. Aging Cell. e13288 |
| Shares BH, Smith CO, Sheu TJ, et al. (2020) Inhibition of the mitochondrial permeability transition improves bone fracture repair. Bone. 115391 |
| Wang Y, Newman MR, Ackun-Farmmer M, et al. (2020) Correction to Fracture-Targeted Delivery of β-Catenin Agonists Peptide-Functionalized Nanoparticles Augments Fracture Healing. Acs Nano |
| Lo CW, Kryvalap Y, Sheu TJ, et al. (2019) Cellular proliferation in mouse and human pancreatic islets is regulated by serpin B13 inhibition and downstream targeting of E-cadherin by cathepsin L. Diabetologia |
| LO C, SHEU T, CZYZYK J. (2018) E-Cadherin Downregulation by Cathepsin L Plays a Role in the Proliferation of Insulin-Producing Cells following Inhibition of Serpin B13 Diabetes. 67: 2126-P |
| Newman MR, Russell SG, Schmitt CS, et al. (2017) Multivalent Presentation of Peptide Targeting Groups Alters Polymer Biodistribution to Target Tissues. Biomacromolecules |
| Wang Y, Newman MR, Ackun-Farmmer M, et al. (2017) Fracture-Targeted Delivery of β-Catenin Agonists via Peptide-Functionalized Nanoparticles Augments Fracture Healing. Acs Nano |
| Beier EE, Sheu TJ, Resseguie EA, et al. (2017) Sclerostin activity plays a key role in the negative effect of glucocorticoid signaling on osteoblast function in mice. Bone Research. 5: 17013 |
| Cheng Q, Tang W, Sheu TJ, et al. (2016) Circulating TGF-β1 levels are negatively correlated with sclerostin levels in early postmenopausal women. Clinica Chimica Acta; International Journal of Clinical Chemistry |
| Beier EE, Holz J, Sheu TJ, et al. (2015) Elevated Lifetime Lead Exposure Impedes Osteoclast Activity and Produces an Increase in Bone Mass in Adolescent Mice. Toxicological Sciences : An Official Journal of the Society of Toxicology |