Sandy Chang

Cancer Genetics University of Texas, Austin, Texas 
"Sandy Chang"


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Ronald A. DePinho post-doc University of Texas (podlevsky) Delete Edit
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Wang Y, Wang X, Flores ER, et al. (2016) Dysfunctional telomeres induce p53-dependent and independent apoptosis to compromise cellular proliferation and inhibit tumor formation. Aging Cell
Rai R, Chen Y, Lei M, et al. (2016) TRF2-RAP1 is required to protect telomeres from engaging in homologous recombination-mediated deletions and fusions. Nature Communications. 7: 10881
Rai R, Chang S. (2015) Monitoring the DNA Damage Response at Dysfunctional Telomeres. Methods in Molecular Biology (Clifton, N.J.). 1343: 175-80
Gupta R, Dong Y, Solomon PD, et al. (2014) Synergistic tumor suppression by combined inhibition of telomerase and CDKN1A. Proceedings of the National Academy of Sciences of the United States of America. 111: E3062-71
Lee Y, Brown EJ, Chang S, et al. (2014) Pot1a prevents telomere dysfunction and ATM-dependent neuronal loss. The Journal of Neuroscience : the Official Journal of the Society For Neuroscience. 34: 7836-44
Wang Y, Sharpless N, Chang S. (2013) p16(INK4a) protects against dysfunctional telomere-induced ATR-dependent DNA damage responses. The Journal of Clinical Investigation. 123: 4489-501
Wan B, Yin J, Horvath K, et al. (2013) SLX4 assembles a telomere maintenance toolkit by bridging multiple endonucleases with telomeres. Cell Reports. 4: 861-9
Min JN, Tian Y, Xiao Y, et al. (2013) The mINO80 chromatin remodeling complex is required for efficient telomere replication and maintenance of genome stability. Cell Research. 23: 1396-413
Gu P, Chang S. (2013) Functional characterization of human CTC1 mutations reveals novel mechanisms responsible for the pathogenesis of the telomere disease Coats plus. Aging Cell. 12: 1100-9
Chang S. (2013) Cancer chromosomes going to POT1. Nature Genetics. 45: 473-5
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