Andrew Koff
Affiliations: | Molecular Biology | Weill Cornell Medical College, New York, NY, United States |
Area:
Biochemistry, Molecular Biology, Cell BiologyGoogle:
"Andrew Koff"Children
Sign in to add traineeThomas B Nguyen | grad student | Weill Cornell Graduate School of Graduate Science | |
Timothy J. Soos | grad student | 2000 | Weill Cornell Medical College |
Diana M. Gitig | grad student | 2001 | Weill Cornell Medical College |
Roshni Bhakta | grad student | 2005 | Weill Cornell Medical College |
Jale Refik-Rogers | grad student | 2006 | Weill Cornell Medical College |
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Publications
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Li Q, Jiang B, Guo J, et al. (2021) INK4 tumor suppressor proteins mediate resistance to CDK4/6 kinase inhibitors. Cancer Discovery |
Dickson MA, Koff A, D'Angelo SP, et al. (2019) Phase 2 study of the CDK4 inhibitor abemaciclib in dedifferentiated liposarcoma. Journal of Clinical Oncology. 37: 11004-11004 |
Klein ME, Dickson MA, Antonescu C, et al. (2018) PDLIM7 and CDH18 regulate the turnover of MDM2 during CDK4/6 inhibitor therapy-induced senescence. Oncogene |
Klein ME, Kovatcheva M, Davis LE, et al. (2018) CDK4/6 Inhibitors: The Mechanism of Action May Not Be as Simple as Once Thought. Cancer Cell |
Kovatcheva M, Klein ME, Tap WD, et al. (2018) Mechanistic understanding of the role of ATRX in senescence provides new insight for combinatorial therapies with CDK4 inhibitors. Molecular & Cellular Oncology. 5: e1384882 |
Kovatcheva M, Liao W, Klein ME, et al. (2017) ATRX is a regulator of therapy induced senescence in human cells. Nature Communications. 8: 386 |
Dickson MA, Schwartz GK, Keohan ML, et al. (2016) Progression-Free Survival Among Patients With Well-Differentiated or Dedifferentiated Liposarcoma Treated With CDK4 Inhibitor Palbociclib: A Phase 2 Clinical Trial. Jama Oncology |
Kovatcheva M, Liu DD, Dickson MA, et al. (2015) MDM2 turnover and expression of ATRX determine the choice between quiescence and senescence in response to CDK4 inhibition. Oncotarget. 6: 8226-43 |
Raheja R, Liu Y, Hukkelhoven E, et al. (2014) The ability of TRIM3 to induce growth arrest depends on RING-dependent E3 ligase activity. The Biochemical Journal. 458: 537-45 |
Liu Y, Raheja R, Yeh N, et al. (2014) TRIM3, a tumor suppressor linked to regulation of p21 Waf1/Cip1 Oncogene. 33: 308-315 |