Lawrence Pfeffer

Affiliations: 
University of Tennessee Health Science Center, Memphis, TN, United States 
Area:
Pathology, Cell Biology, Molecular Biology
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"Lawrence Pfeffer"
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Publications

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Johnson DL, Kumar R, Kakhniashvili D, et al. (2023) Ccr4-Not ubiquitin ligase signaling regulates ribosomal protein homeostasis and inhibits 40S ribosomal autophagy. Biorxiv : the Preprint Server For Biology
Laribee RN, Boucher AB, Madireddy S, et al. (2023) The STAT3-Regulated Autophagy Pathway in Glioblastoma. Pharmaceuticals (Basel, Switzerland). 16
Antonia RJ, Karelehto E, Toriguchi K, et al. (2022) STAT3 regulates inflammatory cytokine production downstream of TNFR1 by inducing expression of TNFAIP3/A20. Journal of Cellular and Molecular Medicine. 26: 4591-4601
Bhattacharya S, Yin J, Yang C, et al. (2022) STAT3 suppresses the AMPKα/ULK1-dependent induction of autophagy in glioblastoma cells. Journal of Cellular and Molecular Medicine
Wang Y, Yang C, Sims MM, et al. (2022) SS-4 is a highly selective small molecule inhibitor of STAT3 tyrosine phosphorylation that potently inhibits GBM tumorigenesis in vitro and in vivo. Cancer Letters. 533: 215614
He Y, Yang C, Wang Y, et al. (2021) Novel structural-related analogs of PFI-3 (SRAPs) that target the BRG1 catalytic subunit of the SWI/SNF complex increase the activity of temozolomide in glioblastoma cells. Bioorganic & Medicinal Chemistry. 53: 116533
Yang C, Wang Y, Sims MM, et al. (2021) Targeting the Bromodomain of BRG-1/BRM Subunit of the SWI/SNF Complex Increases the Anticancer Activity of Temozolomide in Glioblastoma. Pharmaceuticals (Basel, Switzerland). 14
Zhao G, Zhang W, Dong P, et al. (2021) EIF5A2 controls ovarian tumor growth and metastasis by promoting epithelial to mesenchymal transition via the TGFβ pathway. Cell & Bioscience. 11: 70
Wang Y, Yang CH, Schultz AP, et al. (2021) Brahma-Related Gene-1 (BRG1) promotes the malignant phenotype of glioblastoma cells. Journal of Cellular and Molecular Medicine
Zhao G, Wang Q, Wu Z, et al. (2019) Ovarian primary and metastatic tumors suppressed by survivin knockout or a novel survivin inhibitor. Molecular Cancer Therapeutics
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